|1.||Schachner, Melitta: 3 articles (04/2009 - 07/2006)|
|2.||Irintchev, Andrey: 2 articles (04/2009 - 07/2006)|
|3.||You, Jian: 1 article (09/2012)|
|4.||Xu, Ru-Xiang: 1 article (09/2012)|
|5.||Zhao, Hai-Lin: 1 article (09/2012)|
|6.||Cai, Ying-Qian: 1 article (09/2012)|
|7.||Jiang, Xiao-Dan: 1 article (09/2012)|
|8.||Zhang, Mao-Ying: 1 article (09/2012)|
|9.||Xu, Zhi-Min: 1 article (09/2012)|
|10.||Guo, Yang: 1 article (09/2012)|
06/01/1999 - "Tenascin-R and C in multiple sclerosis lesions: relevance to extracellular matrix remodelling."
06/01/1999 - "In the present study the distribution of the inhibitory extracellular molecules tenascin-R (TN-R) and tenascin- C (TN-C) was examined by immunocytochemistry during evolution of the multiple sclerosis (MS) lesion, in which astrogliosis is a prominent feature. "
|2.||Craniocerebral Trauma (Head Injury)
01/01/2008 - "In this study, we employed a recombinant DNA vaccine encoding the myelin-derived inhibitors NogoA, myelin-associated glycoprotein (MAG) and tenascin-R (TnR), so as to effect the production of antibodies against these myelin-related antigens, in a rodent head injury model and ascertained its potential for promoting axonal plasticity and functional recovery. "
|3.||Spinal Cord Injuries (Spinal Cord Injury)
09/13/2012 - "Tenascin-R (TN-R) is a neural specific protein and an important molecule involved in inhibition of axonal regeneration after spinal cord injury (SCI). "
09/13/2012 - "Passive immunization with tenascin-R (TN-R) polyclonal antibody promotes axonal regeneration and functional recovery after spinal cord injury in rats."
07/26/2006 - "Tenascin-R restricts posttraumatic remodeling of motoneuron innervation and functional recovery after spinal cord injury in adult mice."
04/01/2009 - "Enhanced H-reflex responses at frequencies between 0.1 and 5 Hz were also observed in mice deficient in the extracellular matrix glycoprotein tenascin-R and the adhesion molecule close homolog of L1, mice previously shown to have better motor recovery after spinal cord injury than wild-type littermates. "
10/01/2002 - "F3/contactin mediates neuron-glial contacts through its association with extracellular matrix components (tenascin-R, tenascin-C) and RPTPbeta/phosphacan, influencing axonal growth and fasciculation. "
09/01/1991 - "Antibodies to restrictin do not interfere with the fasciculation of retinal axons and the isolated restrictin does not stimulate the outgrowth of axons. "
04/01/2009 - "We investigated the role of the extracellular matrix glycoprotein tenascin-R (TNR) in formation of a hyperexcitable network in the kindling model of epilepsy. "
01/01/2004 - "Mice deficient for the extracellular matrix glycoprotein tenascin-r show physiological and structural hallmarks of increased hippocampal excitability, but no increased susceptibility to seizures in the pilocarpine model of epilepsy."
|3.||Myelin-Associated Glycoprotein (S-MAG)
|4.||Recombinant DNA (Recombinant DNA Research)
|10.||Histone Deacetylase 2