|1.||Krieter, Philip A: 1 article (06/2007)|
|2.||Golembiowska, Krystyna: 1 article (06/2007)|
|3.||Nikiforuk, Agnieszka: 1 article (06/2007)|
|4.||Popik, Piotr: 1 article (06/2007)|
|5.||Tam, Eyal: 1 article (06/2007)|
|6.||Krawczyk, Martyna: 1 article (06/2007)|
|7.||Janowsky, Aaron: 1 article (06/2007)|
|8.||Kowalska, Magdalena: 1 article (06/2007)|
|9.||Skolnick, Phil: 1 article (06/2007)|
|10.||Basile, Anthony S: 1 article (06/2007)|
05/01/1993 - "Bonding durability of photocured phenyl-P in TEGDMA to smear layer-retained bovine dentin."
11/01/2001 - "The purpose of this study was to examine the effect of 2-methacryloyloxyethyl phenyl phosphoric acid (Phenyl-P)/2-hydroxyethyl methacrylate (HEMA) acetone-based primer on moist dentin surfaces that were preconditioned with ethylenediaminetetraacetate (EDTA) to remove the smear layer. "
05/01/1999 - "In a model of persistent chemical pain, the phenyl-p-quinone-induced abdominal constriction assay, 5'd-5IT (ED50 value = 1.5 micromol/kg, SC) and morphine (ED50 value = 3.0 micromol/kg, SC) dose dependently reduced nociception. "
06/01/2007 - "Bicifadine potently suppressed pain responses in both the Randall-Selitto and kaolin models of acute inflammatory pain and in the phenyl-p-quinone-induced and colonic distension models of persistent visceral pain. "
09/28/2006 - "The analgesic and anti-hyperalgesic effects of cannabinoid- and vanilloid-like compounds, plus the fatty acid amide hydrolase (FAAH) inhibitor Cyclohexylcarbamic acid 3'-carbamoyl-biphenyl-3-yl ester (URB597), and acetaminophen, were evaluated in the phenyl-p-quinone (PPQ) pain model, using different routes of administration in combination with opioid and cannabinoid receptor antagonists. "
12/01/1995 - "The compounds tested showed low toxicity and a pharmacological profile similar to other NSAI substances on reducing the edema induced by carrageenan and contractions induced by phenyl-p-quinone; the most active compounds were 1 and 2. In the same way and as expected with these types of substances, the bleeding time increased. "
|1.||Edetic Acid (EDTA)
|3.||2-methacryloyloxyethyl phenyl phosphoric acid
|4.||hydroxyethyl methacrylate (HEMA)
|6.||Cannabinoid Receptors (Cannabinoid Receptor)
|7.||Morphine (MS Contin)