|1.||Koch, Alisa E: 2 articles (10/2014 - 08/2006)|
|2.||Mueller, Michael D: 2 articles (01/2006 - 03/2003)|
|3.||Tak, Paul P: 1 article (10/2014)|
|4.||Baeten, Dominique L: 1 article (10/2014)|
|5.||Isozaki, Takeo: 1 article (10/2014)|
|6.||Yoshida, Ken: 1 article (10/2014)|
|7.||Amin, M Asif: 1 article (10/2014)|
|8.||Korchynskyi, Olexandr: 1 article (10/2014)|
|9.||Ruth, Jeffrey H: 1 article (10/2014)|
|10.||Campbell, Phillip L: 1 article (10/2014)|
10/01/2014 - "To examine whether the citrullinated chemokines epithelial neutrophil-activating peptide 78 (ENA-78)/CXCL5, macrophage inflammatory protein 1α/CCL3, and monocyte chemotactic protein 1/CCL2 are detected in the biologic fluid of patients with rheumatoid arthritis (RA), and if so, to determine the biologic activities of these chemokines. "
08/01/2006 - "To evaluate the efficacy of epigallocatechin-3-gallate (EGCG), a potent antiinflammatory molecule, in regulating interleukin-1beta (IL-1beta)-induced production of the chemokines RANTES (CCL5), monocyte chemoattractant protein 1 (MCP-1/CCL2), epithelial neutrophil-activating peptide 78 (ENA-78/CXCL5), growth-regulated oncogene alpha (GROalpha/CXCL1), and matrix metalloproteinase 2 (MMP-2) activity in rheumatoid arthritis (RA) synovial fibroblasts. "
07/01/2014 - "The combination of HB-EGF (heparin-binding EGF-like growth factor) and CXCL5 (CXCL5/epithelial neutrophil-activating peptide-78) produced a strong synergistic effect on cancer proliferation, epithelial-mesenchymal transition, migration and invasion. "
08/01/2008 - "Tumors from MyD88-KD or TLR5-KD cells revealed the reduced production of neutrophil attracting chemokines (epithelial cell-derived neutrophil-activating peptide-78, macrophage-inflammatory protein alpha, and interleukin-8). "
04/15/1992 - "Peptide 74 and the control peptide 78, which contains a single substitution of serine for the "critical" cysteine residue, were added at 30 microM concentrations to the upper compartment of the Boyden chamber in the chemoinvasion assay using HT1080 and A2058 human tumor cells. "
|3.||Idiopathic Pulmonary Fibrosis
09/01/2005 - "Epithelial neutrophil-activating peptide 78 (ENA-78) and interferon gamma-inducible protein 10 (IP10) belong to the CXC chemokine family and are considered to be important factors in idiopathic pulmonary fibrosis (IPF). "
10/20/2009 - "To examine whether there was an association between epithelial neutrophil activating peptide 78 (ENA-78), interferon-inducible protein 10 (IP-10), vascular endothelial growth factor (VEGF) polymorphism and Chinese patients with idiopathic pulmonary fibrosis (IPF). "
08/01/2005 - "Evaluation of the cytokines interleukin 8 and epithelial neutrophil activating peptide 78 as indicators of inflammation in prostatic secretions."
12/20/2000 - "Evaluation of the cytokines interleukin 8 and epithelial neutrophil activating peptide 78 as indicators of inflammation in prostatic secretions."
12/20/2000 - "To provide objective parameters of inflammation, we measured the cytokines interleukin 8 (IL-8) and epithelial neutrophil activating peptide 78 (ENA-78) in EPS of healthy men, men with benign prostatic hyperplasia (BPH), men with bacterial prostatitis (BP), and men with chronic prostatitis/CPPS. "
01/01/2006 - "Epithelial neutrophil-activating peptide 78 levels were significantly higher in follicular fluid from endometriosis patients than from controls (p = 0.008). "
03/01/2003 - "Epithelial neutrophil-activating peptide 78 may play an important role in the pathogenesis of endometriosis."
03/01/2003 - "To investigate the presence of epithelial neutrophil-activating peptide 78 (ENA-78) in peritoneal fluid of women with and without endometriosis and to identify the cells that produce this inflammatory protein. "
03/01/2003 - "Epithelial neutrophil-activating peptide 78 concentrations are elevated in the peritoneal fluid of women with endometriosis."
01/01/2006 - "Increased follicular fluid levels of epithelial neutrophil-activating peptide 78, tumor necrosis factor-alpha and interleukin-6 indicate that these cytokines may influence oocyte quality and fecundability of women with endometriosis by deteriorating the microenvironment in the human follicle."
|2.||Interleukin-1beta (Interleukin 1 beta)
|4.||Chemokine CCL2 (Monocyte Chemoattractant Protein 1)
|5.||Matrix Metalloproteinase 2 (Gelatinase A)
|6.||epigallocatechin gallate (epigallocatechin-3-gallate)
|7.||Interleukin-8 (Interleukin 8)
|10.||Interferon-gamma (Interferon, gamma)