|1.||Vanin, A F: 6 articles (01/2015 - 05/2007)|
|2.||Tamaki, Toshiaki: 2 articles (02/2010 - 11/2008)|
|3.||Ishizawa, Keisuke: 2 articles (02/2010 - 11/2008)|
|4.||Tsuchiya, Koichiro: 2 articles (02/2010 - 11/2008)|
|5.||Tomita, Shuhei: 2 articles (02/2010 - 11/2008)|
|6.||Pisarenko, O I: 2 articles (05/2009 - 01/2009)|
|7.||Studneva, I M: 2 articles (05/2009 - 01/2009)|
|8.||Gow, Andrew J: 2 articles (01/2007 - 10/2005)|
|9.||Shields, Howard: 2 articles (11/2004 - 07/2003)|
|10.||Lockamy, Virginia L: 2 articles (11/2004 - 07/2003)|
06/05/1992 - "This report demonstrates that iron-nitrosyl EPR signals similar to those observed in macrophages and during sepsis are present in the blood and in the grafted tissue of rats during the rejection of allogeneic (but not syngeneic) heart grafts. "
06/05/1992 - "In cytotoxic activated macrophages, NO production results in a characteristic pattern of alteration of iron-containing enzyme function that is mimicked by exposure to NO. Electron paramagnetic resonance (EPR) studies have shown the formation of iron-nitrosyl species during macrophage activation and also during sepsis, indicating that alteration of iron-containing protein function may be the result of the well-documented tendency of NO to bind to metal ions. "
08/05/1996 - "In the present study, it was found that (1) injection of the NO donor dinitrosyl iron complex (DNIC) into rats results in accumulation of HSP70 in the heart; (2) heat shock is accompanied by increased generation of NO (EPR assay) and HSP70 accumulation in cultured cells; (3) DNIC induces HSP70 accumulation in cultured cells not exposed to heat shock."
02/01/2014 - "Concomitantly, the concentrations of S-nitroso and iron-nitrosyl compounds increased, showing that nitrite was used to produce NO and to S-nitrosate cellular molecules during anoxia. "
02/01/2014 - "Crucian carp appear to exploit this mechanism by upregulating nitrite and other nitrite/NO metabolites (S-nitroso and iron-nitrosyl compounds) in several tissues when exposed to anoxia. "
01/01/1999 - "Dinitrosyl iron complex (DNIC, 200 microg/kg, single dose, iv), a NO donor, decreases the resistance of animals to acute hypoxia by 30%. "
10/01/2012 - "In anoxia, nitrite is reduced to NO, with significant formation of iron-nitrosyl proteins and S-nitrosothiols. "
03/01/2007 - "In conclusion, a significant increase in plasma concentrations of NO metabolites and a modest increase in iron nitrosyl Hb levels were found, suggesting increased luminal consumption caused by erythrocytosis and further suggesting that hypoxemia might activate nonoxidative NO metabolic pathways and enhance tissue oxygen delivery."
02/15/1996 - "K562 cells did not produce any appreciable levels of NO, but they were targeted by RNIs released from the cytokine-stimulated vascular cells, as demonstrated by electron paramagnetic resonance spectrometry, which showed formation of nonheme iron-nitrosyl complexes in the tumor cells. "
02/18/1994 - "We observed that syngeneic murine tumors exhibited EPR signals related to iron-nitrosyl complex formation. "
01/01/2015 - "The selective accumulation of DNICs as well as iron nitrosyl complexes of heme-containing proteins in tumors were detected by EPR method. "
01/01/2013 - "However, ingested sodium nitrite increased fecal nitroso-compounds level, but their fecal concentration and their nature (iron-nitrosyl) would probably not be associated with an increased risk of cancer. "
03/01/2008 - "The addition of dinitrosyl iron complex resulted in blood flow recovery in occlusive vessels and prevented ischemia and necrosis of tissues. "
01/01/2009 - "Effects of exogenous NO donor--dinitrosyl iron complex with reduced glutathione (DNIC-GS) on functional recovery of isolated perfused rat heart subjected to global ischemia and reperfusion have been studied. "
|4.||Glutathione (Reduced Glutathione)
|5.||Nitric Oxide (Nitrogen Monoxide)
|7.||NG-Nitroarginine Methyl Ester (L-NAME)
|8.||Proteins (Proteins, Gene)