|1.||Okada, Masato: 3 articles (05/2011 - 03/2007)|
|2.||Sanjay, Archana: 3 articles (01/2011 - 04/2004)|
|3.||Defilippi, Paola: 2 articles (01/2013 - 03/2004)|
|4.||Cabodi, Sara: 2 articles (01/2013 - 03/2004)|
|5.||Turco, Emilia: 2 articles (01/2013 - 03/2004)|
|6.||Chhikara, Bhupender S: 2 articles (01/2012 - 06/2011)|
|7.||Kumar, Anil: 2 articles (01/2012 - 06/2011)|
|8.||Tiwari, Rakesh: 2 articles (01/2012 - 06/2011)|
|9.||Rao, V Kameshwara: 2 articles (01/2012 - 06/2011)|
|10.||Shirazi, Amir Nasrolahi: 2 articles (01/2012 - 06/2011)|
01/22/2010 - "The nonreceptor protein-tyrosine kinase c-Src is frequently overexpressed and/or activated in a variety of cancers, including those of the breast. "
10/01/2006 - "The increased expression of c-Src kinase and pStat3 expression could play a role in the invasiveness of group 2 tumors. "
10/01/2006 - "Group 2 (invasive) RB showed high c-Src kinase immunoreactivity (Allred score 6-8) in 22/22 tumors and high pStat3 (Allred score 6-8) in 19/22 tumors. "
10/01/2006 - "Group 1 (non-invasive) RB showed intermediate c-Src kinase immunoreactivity (Allred score 4-5) in 14/18 tumors and low immunoreactivity (Allred score 2-3) in 4/18 tumors. "
10/01/2006 - "The immunoreactivity of c-Src kinase and pStat3 was compared in the two groups of tumors. "
|3.||Bone Diseases (Bone Disease)
|4.||Breast Neoplasms (Breast Cancer)
09/01/2007 - "These results indicate a crucial role for c-Src kinase in the survival of ER-positive breast cancer cells only when ER function is blocked. "
11/19/2010 - "In this study, we investigated hyaluronan (HA)-induced CD44 (a primary HA receptor) interaction with c-Src kinase and the transcriptional factor, Twist, in breast tumor cells (MDA-MB-231 cells). "
03/19/2004 - "In this study we report that OPN induces alpha(v)beta(3) integrin-mediated c-Src kinase activity in both highly invasive (MDA-MB-231) and low invasive (MCF-7) breast cancer cells. "
12/29/2000 - "Our findings indicate that PTP1B is the primary protein-tyrosine phosphatase capable of dephosphorylating c-Src in several human breast cancer cell lines and suggests a regulatory role for PTP1B in the control of c-Src kinase activity."
04/01/2011 - "HCB is a tumor cocarcinogen in rat mammary gland and an inducer of cell proliferation and c-Src kinase activity in MCF-7 breast cancer cells. "
09/01/2007 - "These data suggest that post-translational modification of Ca(2+) channels during inflammation, possibly nitrotyrosylation, prevents c-Src kinase regulation resulting in decreased Ca(2+) influx."
09/01/2007 - "Neither the Ca(2+) channel Ca(v)1.2b, gene expression, nor the c-Src kinase activity was altered by inflammation. "
02/01/2004 - "C-terminal SRC kinase controls acute inflammation and granulocyte adhesion."
03/01/2010 - "Tyrosine nitration of Ca(2+) channels attenuates interactions with c-Src kinase, decreasing Ca(2+) channel currents and smooth muscle contraction during colonic inflammation. "
12/01/2004 - "These studies suggest that decreased calcium channel currents following colonic inflammation are not due to decreased expression but may result from altered regulation by the non-receptor cellular tyrosine kinase, c-src kinase."
|1.||Hyaluronic Acid (Hyaluronan)
|2.||Protein-Tyrosine Kinases (Tyrosine Kinase)
|3.||Proteins (Proteins, Gene)
|4.||Integrin beta3 (GPIIIa)
|5.||Calcium Channels (Calcium Channel)
|9.||Carrier Proteins (Binding Protein)
|1.||Heterologous Transplantation (Xenotransplantation)