|1.||Kobayashi, Motohiro: 4 articles (05/2009 - 12/2004)|
|2.||Nakayama, Jun: 4 articles (05/2009 - 12/2004)|
|3.||Fukuda, Minoru: 4 articles (05/2009 - 12/2004)|
|4.||Lee, Heeseob: 2 articles (05/2009 - 01/2009)|
|5.||Sebestyén, Tímea: 1 article (10/2014)|
|6.||Ladányi, Andrea: 1 article (10/2014)|
|7.||Tóth, Erika: 1 article (10/2014)|
|8.||Somlai, Beáta: 1 article (10/2014)|
|9.||Tímár, József: 1 article (10/2014)|
|10.||Liszkay, Gabriella: 1 article (10/2014)|
10/01/2014 - "MECA-79(+) HEV-like venules were observed in the neighborhood of the follicles in the majority of cases, however, their presence was not confined to tumors hosting ectopic lymphoid structures. "
12/01/1991 - "Furthermore, tumors expressed human LFA-1/ICAM-1 and the tumor blood vessels strongly expressed murine ICAM-1, but not MECA 79. "
09/01/2011 - "In this article, we report the presence of MECA 79(+) blood vessels displaying all the phenotypic characteristics of HEVs in most of the 319 human primary solid tumors, including melanomas, breast, ovarian, colon, and lung carcinomas, analyzed. "
06/01/1995 - "Tumor vasculature showed signs of activation in each patient on the basis of elevated levels of intercellular adhesion molecule-1, E-selectin, vascular cell adhesion molecule-1, and/or peripheral lymph node addressin (PNAd). "
12/01/1993 - "The extravasation of blood-borne lymphocytes into organized lymphoid tissues and sites of chronic inflammation is directed in part by interactions of lymphocyte surface adhesion molecules, known as homing receptors, with tissue-selective endothelial ligands called vascular addressins. "
12/01/1994 - "Because MECA 79 stains HEV-like vessels at various sites of inflammation, recognition by L-selectin of ligands outside of secondary lymphoid organs may depend on sulfation. "
05/01/2001 - "The adhesiveness of HEV ligands for L-selectin and alpha4beta7 integrin (ie, peripheral lymph node addressin and mucosal addressin cell adhesion molecule-1) also increased during WBH or febrile responses associated with lipopolysaccharide-induced or turpentine-induced inflammation. "
11/01/1997 - "pylori-associated follicular gastritis and MALT lymphomas high endothelial venules coexpressed mucosal addressin cell adhesion molecule-1 and peripheral lymph node addressin. "
01/01/2009 - "In chronic gastritis, lymphocytes infiltrate the lamina propria, and such infiltration is facilitated by 6-sulfo sialyl Lewis X-capped O-glycans, peripheral lymph node addressin (PNAd), on high endothelial venule (HEV)-like vessels. "
05/01/2009 - "In chronic gastritis, lymphocytes infiltrate the lamina propria, and such infiltration is facilitated by the interaction between L-selectin on lymphocytes and peripheral lymph node addressin (PNAd), which contains 6-sulfo sialyl Lewis X-capped O-glycans, on high endothelial venule (HEV)-like vessels. "
02/01/1992 - "Human, human/mouse CNS lymphomas, normal human, and mouse brains showed no staining with either HECA 452 or MECA 79.(ABSTRACT TRUNCATED AT 250 WORDS)"
04/01/1990 - "Here we demonstrate that Peyer's patch high endothelial venules express a low but functional level of peripheral lymph node addressin (MECA-79) that can be recognized by lymphomas expressing the peripheral lymph node homing receptor (MEL-14 antigen).(ABSTRACT TRUNCATED AT 250 WORDS)"
02/15/2007 - "Differential expression of homing receptors and vascular addressins in tonsils and draining lymph nodes: Effect of Brucella infection in sheep."
12/21/2004 - "pylori infection is highly correlated with de novo synthesis of peripheral lymph node addressin (PNAd) presented on high-endothelial venule (HEV)-like vessels. "
|3.||Intercellular Adhesion Molecule-1 (Intercellular Adhesion Molecule 1)
|4.||sialyl- 2- 3- (6'- sulfo)galactosyl- 1- 4- (fucopyranosyl- 1- 3)- N- acetylglucosamine
|5.||Lymphocyte Homing Receptors
|8.||Vascular Cell Adhesion Molecule-1 (Vascular Cell Adhesion Molecule 1)