|1.||Dong, Zigang: 3 articles (02/2014 - 09/2007)|
|2.||Cho, Yong-Yeon: 3 articles (02/2014 - 09/2007)|
|3.||Lee, Mee-Hyun: 2 articles (02/2014 - 01/2011)|
|4.||Shoemaker, Robert H: 2 articles (09/2011 - 09/2006)|
|5.||Yao, Ke: 2 articles (01/2011 - 09/2007)|
|6.||Bode, Ann M: 2 articles (01/2011 - 09/2007)|
|7.||Lee, Cheol-Jung: 1 article (02/2014)|
|8.||Lee, Ji Young: 1 article (02/2014)|
|9.||Lee, Hyeong-Kyu: 1 article (02/2014)|
|10.||Li, Yan: 1 article (02/2014)|
07/15/2004 - "In the present study, we show that VV infection provoked a sustained activation of both ERK1/2 and RSK2 (ribosomal S6 kinase 2). "
03/01/2009 - "Here, we report that the ribosomal protein S6 kinase alpha 3 (RSK2), which is activated through the "classical" mitogen-activated protein kinase pathway, plays a role in innate immune responses to influenza virus infection. "
|3.||Coffin-Lowry Syndrome (Syndrome, Coffin-Lowry)
10/01/2009 - "Mutations in Ribosomal s6 kinase 2 (Rsk2) are associated with severe neuronal dysfunction in Coffin-Lowry syndrome (CLS) patients, flies and mice. "
12/01/2003 - "Two novel mutations of the ribosomal S6 kinase 2 gene (also known as RSK2) have been identified in two unrelated patients with Coffin-Lowry syndrome. "
10/01/1999 - "An unreported missense mutation of the ribosomal S6 kinase 2 (RSK2) gene has been identified in two male sibs with a mild form of Coffin-Lowry syndrome (CLS) inherited from their healthy mother. "
10/01/2001 - "Coffin-Lowry syndrome is a rare X-linked, semi-dominant mental retardation syndrome resulting from mutations of the ribosomal S6 kinase 2 (RSK2) gene. "
12/11/2013 - "We show here that neurons cultured from mice lacking ribosomal S6 kinase 2 (Rsk2), a model for the Coffin-Lowry syndrome (CLS), exhibit a significant delay in growth in a similar way to that shown by neurons cultured from phospholipase D1 (Pld1) knock-out mice. "
09/01/2011 - "It was found that a small subset of compounds show both potent inhibition of RSK2 kinase activity and tumor cell growth in vitro. "
09/01/2007 - "Ribosomal S6 kinase 2 is a key regulator in tumor promoter induced cell transformation."
02/01/2014 - "Mitogen-activated protein kinases play a key role in cell proliferation, cell cycle progression and cell transformation, and activated Ras/extracellular signal-regulated kinases (ERKs)/ribosomal S6 kinase 2 (RSK2) signaling pathways have been widely identified in many solid tumors. "
09/01/2006 - "Ribosomal S6 kinase 2 (RSK2) is a serine/threonine kinase that plays a role in human cancer and Coffin-Lowry syndrome and is comprised of two nonidentical kinase domains, each domain with its own ATP-binding site. "
09/01/2011 - "Our results in estrogen-unresponsive fibroblasts, ribosomal S6 kinase 2 kinase (RSK2) knockout cells, and estrogen receptor (ER)-deficient breast tumor cells show that kurarinone can inhibit tumor cell proliferation and selectively block nuclear NFκB transactivation of specific target genes such as IL6, cyclin D1, SOD2 but not TNFAIP2. "
|1.||Ribosomal Protein S6 Kinases (S6 Kinase)
|2.||Ribosomal Proteins (Ribosomal Protein)
|4.||Protein Kinases (Protein Kinase)
|6.||Protein Phosphatase 1
|7.||Extracellular Signal-Regulated MAP Kinases (Extracellular Signal Regulated Kinases)
|9.||LDL-Receptor Related Protein 1 (LDL-Receptor Related Protein)
|10.||Mitogen-Activated Protein Kinases