|1.||Fang, Xiao Yi: 1 article (08/2010)|
|2.||Lin, Ni Yang: 1 article (08/2010)|
|3.||Li, Yu Guang: 1 article (08/2010)|
|4.||Huang, Ning: 1 article (01/2009)|
|5.||Ashrafpour, Homa: 1 article (01/2009)|
|6.||Cahoon, Neil J: 1 article (01/2009)|
|7.||Pang, Cho Y: 1 article (01/2009)|
|8.||Neligan, Peter C: 1 article (01/2009)|
|9.||Jindal, Kunaal: 1 article (01/2009)|
|10.||Moses, Michael A: 1 article (01/2009)|
|1.||Wounds and Injuries (Trauma)
09/01/2006 - "Rats given PS alone exhibited hepatic fibrosis as well as increases in the number of the alpha-SMA-positive HSCs and NHE-1 protein expression in HSCs and hepatocytes, all of which were suppressed by SPL. "
09/01/2006 - "PS-induced hepatic fibrosis together with HSC activation and NHE-1 protein expression occurs via MRs, and SPL ameliorates hepatic fibrosis presumably via the inhibition of HSC activation and NHE-1 protein expression in PS-induced liver injuries. "
|3.||Pathologic Constriction (Stenosis)
08/01/2010 - "This study was designed to explore the effects of amiloride, a Na+-H+ exchange (NHE) inhibitor, on vessel stenosis by observing the expression of NHE-1 protein in vascular smooth muscle (VSM) after balloon injury and the effects of amiloride on VSM cell proliferation, migration, and excretion of extracellular matrices (ECMs). "
04/01/2005 - "Our results showed that simultaneous intracellular Na(+) and Ca(2+) overloads as well as an increase of NHE-1 protein level took place during the development of necrosis and hypertrophy in the CMH. "
04/01/2005 - "Treatment of 30-day-old CMHs during the development of necrosis and in the absence of hypertrophy with the specific NHE-1 inhibitor EMD87580 (EMD) for 50 days significantly prevented the increase of NHE-1 protein level and intracellular Na(+) and Ca(2+) overloads as well as necrosis. "
|2.||glucuronyl glucosamine glycan sulfate (Vessel)
|3.||Messenger RNA (mRNA)
|4.||Sodium-Potassium-Exchanging ATPase (Sodium, Potassium ATPase)
|5.||N- (2- methyl- 4,5- bis(methylsulfonyl)benzoyl)guanidine