superoxide dismutase-poly(styrene co-maleic acid butyl ester)

05/01/1993 - "The protective effects of SM-SOD on regional cerebral blood flow, intracranial pressure, cardiac index, vascular permeability, and neurological outcome were investigated using a model of global brain ischemia in dogs. "

01/01/1995 - "Administration of SM-SOD immediately after inducing burn injury also significantly inhibited the increase in CL. "
07/01/1993 - "Intravenous administration of SM-SOD (10 mg/kg) 30 minutes before the burn injury significantly inhibited the increase of plasma lipid peroxides and the occurrence of intravascular hemolysis. "
09/01/1994 - "To determine whether superoxide radicals may influence the pathogenesis of burn injury, we investigated the effects of a long-acting, site-directed superoxide dismutase derivative (SM-SOD) on the rats subjected to burn shock. "
09/01/1994 - "Also, administration of SM-SOD markedly inhibited the increase in the levels of thiobarbituric acid reactive metabolites, such as lipid peroxides in the plasma, lung and kidney, and the decrease in plasma protein levels particularly at the early stage of burn injury. "
07/01/1993 - "To test whether superoxide radicals play critical roles in burn injury, the effect of a superoxide dismutase (SOD) derivative (SM-SOD) was tested on intravascular hemolysis and renal tubular injury in rats subjected to burn shock. "

09/01/1994 - "To determine whether superoxide radicals may influence the pathogenesis of burn injury, we investigated the effects of a long-acting, site-directed superoxide dismutase derivative (SM-SOD) on the rats subjected to burn shock. "
07/01/1993 - "To test whether superoxide radicals play critical roles in burn injury, the effect of a superoxide dismutase (SOD) derivative (SM-SOD) was tested on intravascular hemolysis and renal tubular injury in rats subjected to burn shock. "

08/08/1989 - "These results suggest that superoxide radical and/or its metabolite(s) would play an important role in the pathogenesis of postischemic reperfusion arrhythmias and that SM-SOD may be useful for decreasing tissue injury in ischemic heart disease."

01/01/1991 - "When the left anterior descending artery was occluded permanently, about 65% of animals died within 30 min predominantly due to irreversible ventricular fibrillation; the motality of animals decreased to 15% by administering SM-SOD either before or after occlusion. "