|1.||Tracey, Irene: 2 articles (03/2013 - 02/2011)|
|2.||Capotorto, John V: 1 article (07/2015)|
|3.||Millington, J Thomas: 1 article (07/2015)|
|4.||Stavosky, James W: 1 article (07/2015)|
|5.||Rogers, Lee C: 1 article (07/2015)|
|6.||DellaCorte, Michael P: 1 article (07/2015)|
|7.||Leknes, Siri: 1 article (03/2013)|
|8.||Biele, Guido: 1 article (03/2013)|
|9.||Snyder, Gregory D: 1 article (03/2013)|
|10.||Berna, Chantal: 1 article (03/2013)|
03/01/2013 - "Furthermore, the change in outcome hedonics correlated with activity in the periacqueductal grey (PAG) of the descending pain modulatory system (DPMS). "
02/23/2011 - "Preclinical evidence suggests that opioid withdrawal induces central sensitization (CS) that is maintained by supraspinal contributions from the descending pain modulatory system (DPMS). "
03/01/1999 - "THP analogs enhanced the activity of brainstem DPMS by the blockade of D2 receptors in the striatum and accumbens nucleus, and sequentially inhibited the inputs of peripheral pain afferent message in spinal cord level."
03/01/1999 - "THP analogs, however, the numbers of FLI neurons induced by formalin-pain in the APAS, such as dorsal horn (mainly laminae I, II, IV-VI) were markedly decreased, while the numbers of FLI neurons in the DPMS, such as periaqueductal gray (PAG) and reticular paragigantocellular lateral nucleus (RPLN) were significantly increased. "
03/01/1999 - "In the formalin-pain group, FLI neurons were mainly distributed in ascending pain afferent system (APAS) and descending pain modulation system (DPMS). "
|2.||Wounds and Injuries (Trauma)
|3.||African Trypanosomiasis (Nagana)
04/01/1990 - "At 24 h, kidneys from rats treated with phenobarbital plus DPMS (1.0 mmol kg-1) exhibited extensive proximal tubular necrosis and numerous glomeruli with thickened membranes. "
04/01/1990 - "Phenobarbital pretreatment did not markedly enhance the functional nephrotoxicity induced by DPMS (0.4 mmol), but tubular necrosis was greater than observed in non-phenobarbital-pretreated rats receiving DPMS (1.0 mmol kg-1). "
04/01/1990 - "BPMS and DPMS (1.0 mmol kg-1) treatment induced mild renal tubular necrosis and thickening of the glomerular membranes. "
|1.||Keratin-7 (Cytokeratin 7)
|2.||Keratin-19 (Keratin 19)
|3.||Hepatocyte Nuclear Factor 6
|4.||Neural Cell Adhesion Molecules (Neural Cell Adhesion Molecule)
|7.||Dolichol Monophosphate Mannose
|8.||1- methyl- 3- (4- (1- methylethyl)phenyl)- 2,5- pyrrolidinedione (BPMS)