|1.||Aoe, Tomohiko: 2 articles (01/2014 - 09/2004)|
|2.||Mimura, Naoya: 2 articles (01/2014 - 09/2004)|
|3.||Koseki, Haruhiko: 2 articles (01/2014 - 09/2004)|
|4.||Kashio, Makiko: 1 article (01/2014)|
|5.||Jin, Hisayo: 1 article (01/2014)|
|6.||Wang, G: 1 article (09/2011)|
|7.||Li, B: 1 article (09/2011)|
|8.||Zhou, L: 1 article (09/2011)|
|9.||Fei, E: 1 article (09/2011)|
|10.||Wang, P: 1 article (09/2011)|
|1.||Neurodegenerative Diseases (Neurodegenerative Disease)
01/01/2014 - "The defect in retrieval of BiP by the KDEL receptor leads to impaired activities in quality control and autophagy, suggesting that functional defects in the ER chaperones may contribute to the late onset of neurodegenerative diseases. "
09/08/2011 - "The KDEL receptor induces autophagy to promote the clearance of neurodegenerative disease-related proteins."
09/12/2000 - "We report immunoelectron microscopy experiments establishing that a Golgi-restricted SNARE, GOS 28, is present in the same population of COPI vesicles as anterograde cargo marked by vesicular stomatitis virus glycoprotein, but is excluded from the COPI vesicles containing retrograde-targeted cargo (marked by KDEL receptor). "
01/06/2009 - "A role for the host coatomer and KDEL receptor in early vaccinia biogenesis."
01/06/2009 - "Whereas coatomer forms COPI vesicles in the host early secretory system, vaccinia formation bypasses this role of coatomer, but instead, depends on coatomer interacting with the host KDEL receptor. "
08/12/2011 - "The analysis of Golgi ultrastructure and membrane trafficking showed that 1) the accumulation of the budding vesicular profiles induced by propanolol was prevented by inhibition of PC synthesis, 2) the density of KDEL receptor-containing punctated structures at the endoplasmic reticulum-Golgi interface correlated with the amount of DAG, and 3) the post-Golgi transport of the yellow fluorescent temperature-sensitive G protein of stomatitis virus and the secretion of a secretory form of HRP were both reduced when DAG was lowered. "
|5.||Dilated Cardiomyopathy (Cardiomyopathy, Congestive)
09/01/2004 - "We found that the mutant KDEL receptor sensitized cells to ER stress and that the mutant mice developed dilated cardiomyopathy. "
09/01/2004 - "Dilated cardiomyopathy caused by aberrant endoplasmic reticulum quality control in mutant KDEL receptor transgenic mice."
09/01/2004 - "These findings suggest that impairment of the KDEL receptor disturbs ER quality control, resulting in accumulation of misfolded proteins in the ER in an in vivo system, and that the dilated cardiomyopathy found in the mutant KDEL receptor transgenic mice is associated with ER stress."
|1.||Proteins (Proteins, Gene)
|2.||Coat Protein Complex I (COPI)
|3.||GTP-Binding Proteins (G-Protein)