|1.||Rosenbauer, Frank: 6 articles (07/2014 - 06/2004)|
|2.||Tenen, Daniel G: 6 articles (08/2008 - 01/2003)|
|3.||Steidl, Ulrich: 4 articles (10/2015 - 01/2006)|
|4.||Ruscetti, Sandra: 3 articles (10/2010 - 06/2006)|
|5.||Wagner, Katharina: 3 articles (11/2006 - 06/2004)|
|6.||Iwasaki, Hiromi: 3 articles (01/2006 - 06/2004)|
|7.||Akashi, Koichi: 3 articles (01/2006 - 06/2004)|
|8.||Will, Britta: 2 articles (10/2015 - 08/2008)|
|9.||Bartholdy, Boris: 2 articles (10/2015 - 08/2008)|
|10.||Yu, J: 2 articles (07/2014 - 01/2014)|
|1.||Acute Erythroblastic Leukemia (Erythroleukemia)
01/29/2010 - "Dysregulated expression of transcription factor PU.1 is strongly associated with Friend virus-induced erythroleukemia. "
02/15/2008 - "Interaction between the homeodomain protein HOXC13 and ETS family transcription factor PU.1 and its implication in the differentiation of murine erythroleukemia cells."
06/01/2006 - "The second stage results from the outgrowth of a rare virus-infected erythroid cell that expresses nonphysiological levels of the myeloid transcription factor PU.1. These cells exhibit a differentiation block and can be grown as murine erythroleukemia (MEL) cell lines. "
02/14/2002 - "The regulation of PU.1 transcription in established erythroleukemia cell lines differed depending upon the level of PU.1 protein present. "
04/15/2001 - "Lineage switch induced by overexpression of Ets family transcription factor PU.1 in murine erythroleukemia cells."
|2.||Acute Myeloid Leukemia (Acute Myelogenous Leukemia)
11/15/2003 - "Mutation analysis of the transcription factor PU.1 in younger adults (16 to 60 years) with acute myeloid leukemia: a study of the AML Study Group Ulm (AMLSG ULM)."
10/01/2015 - "Although loss or near-complete loss of the hematopoietic transcription factor PU.1 induces acute myeloid leukemia (AML) in mice, a similar degree of PU.1 impairment is exceedingly rare in human AML; yet, moderate PU.1 inhibition is common in AML patients. "
01/01/2013 - "Transcription factor PU.1, a member of the ETS family, is a master regulator of myeloid differentiation whose functional disruption is often associated with acute myeloid leukemia (AML). "
11/01/2006 - "Knockdown of the transcription factor PU.1 (encoded by Sfpi1) leads to acute myeloid leukemia (AML) in mice. "
06/01/2004 - "Acute myeloid leukemia induced by graded reduction of a lineage-specific transcription factor, PU.1."
02/28/2015 - "IL-32θ inhibits monocytic differentiation of leukemia cells by attenuating expression of transcription factor PU.1."
07/01/2014 - "Here we show that the transcription factor PU.1 is essential for MLL leukemia and is required for the growth of MLL leukemic cells via the promotion of cell-cycle progression and inhibition of apoptosis. "
04/01/2014 - "Regulation of the hematopoietic transcription factor PU.1, a member of the ETS family, plays a critical role in the development of blood cells and in leukemia. "
08/01/2008 - "The transcription factor PU.1 is an important regulator of hematopoiesis; precise expression levels are critical for normal hematopoietic development and suppression of leukemia. "
04/01/2005 - "Regulation of the hematopoietic transcription factor PU.1 (Spi-1) plays a critical role in the development of white cells, and abnormal expression of PU.1 can lead to leukemia. "
12/01/2015 - "The transcription factor PU.1 promotes alternative macrophage polarization and asthmatic airway inflammation."
05/01/2014 - "Our data provides evidence that miR-155 contributes to the regulation of allergic airway inflammation by modulating T(H)2 responses through the transcription factor PU.1."
06/01/2010 - "The transcription factor PU.1 is required for the development of IL-9-producing T cells and allergic inflammation."
01/01/2008 - "Characterization and expression of the transcription factor PU.1 during LPS-induced inflammation in the rainbow trout (Oncorhynchus mykiss)."
10/01/2010 - "In the second stage, SFFV integration into the Sfpi1 locus activates the myeloid transcription factor PU.1, blocking erythroid cell differentiation, and in conjunction with the loss of p53 tumor suppressor activity, results in the outgrowth of malignant cells. "
01/01/2006 - "These results elucidate how a single transcription factor, PU.1, through the cell context-specific activity of a key cis-regulatory element, affects the development of multiple cell lineages and can induce cancer."
05/01/2008 - "Tumor tissues were CpG hypomethylated and showed a modest elevation of the transcription factor PU.1, an oncogene that is crucial for Friend virus induced erythroleukemia. "
|1.||Interleukin-9 (Interleukin 9)
|2.||Messenger RNA (mRNA)
|3.||Interleukin-9 Receptors (Interleukin 9 Receptor)
|5.||Transcription Factor TFIIB (TFIIB)
|6.||Transcription Factor TFIID (TFIID)
|7.||Erythropoietin Receptors (Erythropoietin Receptor)
|9.||Granulocyte Colony-Stimulating Factor (G-CSF)
|1.||Transplantation (Transplant Recipients)