|1.||Ulbricht, Ulrike: 2 articles (09/2006 - 12/2003)|
|2.||Lamszus, Katrin: 2 articles (09/2006 - 12/2003)|
|3.||Fillbrandt, Regina: 2 articles (09/2006 - 12/2003)|
|4.||Westphal, Manfred: 2 articles (09/2006 - 12/2003)|
|5.||Eckerich, Carmen: 2 articles (09/2006 - 12/2003)|
|6.||Zea, Arnold H: 1 article (04/2009)|
|7.||Manning, Jennifer: 1 article (04/2009)|
|8.||Aviles, Diego H: 1 article (04/2009)|
|9.||Ochoa, Augusto C: 1 article (04/2009)|
|10.||Vehaskari, Matti V: 1 article (04/2009)|
04/22/2003 - "More importantly, the scFv(anti-HER-2/neu)/zeta receptor was functionally active, since it triggered cytokine secretion by the MD.45-HER/zeta cells upon recognition of HER-2/neu-positive (+) tumour cell lines, or primary tumour cells from patients with HER-2/neu(+) cancers. "
09/01/1996 - "OGF and its receptor, zeta (zeta), were detected in transplanted human HT-29 colon tumors. "
07/15/1995 - "Chimeric zeta-receptors direct human natural killer (NK) effector function to permit killing of NK-resistant tumor cells and HIV-infected T lymphocytes."
08/01/2004 - "Chimeric receptor, scFv(anti-HER-2/neu)/zeta receptor, was successfully used to redirect cytotoxic T lymphocyte hybridoma cells to obtain anti-HER-2/neu positive tumor cells, suggesting the feasibility of treatment of breast cancer patients with T cell receptor gene transfer immunotherapy. "
02/01/2006 - "It has been suggested that the release of these putative endogenous ligands of TLRs during cancer progression may cause chronic inflammation leading to the recruitment of myeloid suppressor cells and down-regulation of T-cell and natural killer (NK) cell receptor zeta (zeta) chain resulting in T and NK cell dysfunction. "
|2.||Glioblastoma (Glioblastoma Multiforme)
09/01/2006 - "RNA interference targeting protein tyrosine phosphatase zeta/receptor-type protein tyrosine phosphatase beta suppresses glioblastoma growth in vitro and in vivo."
09/01/2006 - "The protein tyrosine phosphatase zeta/receptor-type protein tyrosine phosphatase beta (PTPzeta/RPTPbeta) and its ligand pleiotrophin (PTN) are overexpressed in human glioblastomas. "
07/22/2005 - "The secreted growth factor pleiotrophin (PTN) promotes glioblastoma migration and proliferation, initiating its oncogenic activities through two cell surface receptors, the protein tyrosine phosphatase receptor zeta (PTPRZ1) and the anaplastic lymphoma kinase (ALK), respectively. "
12/01/2003 - "Using subtractive cloning combined with cDNA array analysis, we previously identified the genes encoding for the protein tyrosine phosphatase zeta/receptor-type protein tyrosine phosphatase beta (PTPzeta/RPTPbeta) and its ligand pleiotrophin (PTN) as overexpressed in human glioblastomas compared to normal brain. "
06/04/2004 - "Non-opioid actions are exerted through different neuronal receptors, e.g., dynorphin hyperalgesia through NMDA receptor, Met-enkephalin induced regulation of cell growth through zeta receptors, pain modulation by nociceptin through ORL-1 or NOP receptors, while BAM22 acts through sensory neuron specific G protein-coupled receptors (SNSR). "
|5.||Nephrotic Syndrome (Syndrome, Nephrotic)
|1.||Protein Tyrosine Phosphatases
|3.||Class 5 Receptor-Like Protein Tyrosine Phosphatases
|4.||Opioid Receptors (Opioid Receptor)
|5.||G-Protein-Coupled Receptors (Receptors, G Protein Coupled)
|6.||Opioid Peptides (Opioid Peptide)
|7.||Complementary DNA (cDNA)
|10.||Cell Surface Receptors