|1.||Ponchet, Michel: 1 article (07/2013)|
|2.||Panabières, Franck: 1 article (07/2013)|
|3.||Minet-Kebdani, Naïma: 1 article (07/2013)|
|4.||Attard, Agnès: 1 article (07/2013)|
|5.||Gourgues, Mathieu: 1 article (07/2013)|
|6.||Kuhn, Marie-Line: 1 article (07/2013)|
|7.||Govetto, Benjamin: 1 article (07/2013)|
|8.||Evangelisti, Edouard: 1 article (07/2013)|
|9.||Sano, Hiroshi: 1 article (12/2007)|
|10.||Chung, Kwi-Mi: 1 article (12/2007)|
12/01/1997 - "Moreover, exogenous application of RNase activity in the extracellular space of leaves led to a reduction of the fungus development by up to 90%, independently of any cryptogein treatment and in the absence of apparent necrosis. "
07/01/1992 - "Cryptogein (CRY), a protein secreted by Phytophthora cryptogea, causes necrosis on tobacco (Nicotiana tabacum) plants at the site of application (the stem or the roots) and also on distant leaves. "
08/15/1989 - "Cryptogein causes visible leaf necrosis starting at about 1 microgram/plant, whereas 50-fold as much capsicein is required for the same reaction. "
03/01/1992 - "On tobacco plants, they could be classified into two classes: a, comprising capsicein and parasiticein (less necrotic), and β, comprising cryptogein and cinnamomin (very toxic with a necrosis threshold of 0.1 μg per leaf). "
08/15/1989 - "The phytopathogenic fungi Phytophthora cryptogea and Phytophthora capsici cause systemic leaf necrosis on their non-host tobacco; in culture they release proteins, called cryptogein and capsicein, which elicit similar necrosis. "
07/01/2013 - "The PSE1 protein facilitated plant infection: it suppressed plant cell death activated by Pseudomonas syringae avirulence gene AvrPto and Phytophthora cryptogea elicitin cryptogein in tobacco and exacerbated disease symptoms upon inoculation of transgenic A. "
02/01/1999 - "parasitica var nicotianae stimulated cryptogein production that coincided with the fast induction of several defense genes at and around the infection sites. "
12/01/1997 - "Stem application of cryptogein leads to the establishment of acquired resistance to subsequent leaf infection with Phytophthora parasitica var nicotianae, the agent of the tobacco black shank disease. "
06/01/1998 - "The promoter responded to wounding but not to oligogalacturonides, fungal glucan, salicylic acid, cryptogein, or pathogen infection. "
12/01/2007 - "Overexpression of NtWIF in tobacco plants enhanced the hypersensitive response (HR) upon tobacco mosaic virus infection and cryptogein treatment, while its silencing by RNAi suppressed such HR. NtWIF contains a specific motif similar to the B3 DNA binding domain, which recognizes the core TGTCTC motif called the auxin-responsive element (ARE). "
|3.||Wounds and Injuries (Trauma)
05/01/1992 - "Using elicitins radioactively labelled in vivo, we have demonstrated that cryptogein and capsicein (i) move from a wound in the stem towards the leaves where they interact directly, (ii) reach their target without undergoing any molecular alteration, (iii) are carried in, and at the same rate as, the sap flow in the xylem, (iv) do not alter the rate of the xylem flow although they are able to provoke drastic damage to the lamina. "
|1.||Phytophthora capsici Alpha-elicitin capsicein protein
|2.||Staphylococcal Protein A (A, Protein)
|3.||Phytophthora cinnamomi cinnamomin
|4.||Salicylic Acid (2 Hydroxybenzoic Acid)
|7.||Proteins (Proteins, Gene)
|8.||Indoleacetic Acids (Auxin)
|9.||DNA (Deoxyribonucleic Acid)