|1.||Teoh, Melissa L T: 2 articles (07/2013 - 05/2005)|
|2.||Fisher, Aron B: 2 articles (04/2008 - 12/2005)|
|3.||Chatterjee, Shampa: 2 articles (04/2008 - 12/2005)|
|4.||Zhang, Qunwei: 2 articles (04/2008 - 12/2005)|
|5.||Chan, Pak H: 2 articles (03/2007 - 03/2002)|
|6.||Sun, Li: 1 article (08/2014)|
|7.||Wolferts, Guido: 1 article (08/2014)|
|8.||Veltkamp, Roland: 1 article (08/2014)|
|9.||Inoue, Kentaro: 1 article (01/2014)|
|10.||Terami, Takahiro: 1 article (01/2014)|
04/01/2013 - "Pretreatment with RG extract followed by ischemia/reperfusion (I/R) resulted in significant reduction of oxidized hydroethidine signals in ischemic areas. "
01/01/2013 - "An increase in fluorescence was observed during the onset of ischemia in hearts perfused with a solution of hydroethidine, a fluorescent dye sensitive to intracellular O2(•-). "
02/01/1997 - "Surface fluorometry with 40 microM hydroethidine (HE) as a probe was used to detect oxidant generation in isolated, ventilated rat lungs during lung ischemia. "
01/01/2010 - "NSC23766 also attenuated "in situ" O(2)(-) production in the hippocampus after ischemia/reperfusion, as determined by fluorescent oxidized hydroethidine staining. "
01/01/1998 - "Sod2 -/+ mice demonstrated a prominent increase in O2- production under normal physiological conditions and in ischemia, as evidenced by specific oxidation of a fluorescent probe, hydroethidine, reflecting decreased activity of Mn-SOD. "
01/01/2005 - "In the present study, we exposed SHSY5Y neuroblastoma cells to neurotoxic beta amyloid peptides (Abeta) and oxygen glucose deprivation (OGD) to investigate the neuroprotective effect of 10 microM CoQ10 by measuring (i) cell viability by the MTT assay, (ii) opening of the mitochondrial permeability transition pore via the fluorescence intensity of calcein-AM, and (iii) superoxide anion concentration by hydroethidine. "
01/01/2008 - "Exposure to hypoxia caused the generation of intracellular superoxide as evidenced by intense staining of arterial muscle with the fluorescent probe hydroethidine, by quantitation using fluorescent HPLC analysis, and by attenuation of the hypoxia-induced activation of the K(Ca) channel current by superoxide dismutation. "
12/01/2006 - "Exposure of astrocytes to hypoxia was associated with generation of superoxide as detected by staining of cells with the intracellular superoxide detection probe hydroethidine (HE), attenuation of the hypoxia-induced activation of unitary K(Ca) channel currents by superoxide dismutation with tempol, and as quantitated by high-pressure liquid chromatography/fluorescence assay using HE as a probe. "
11/01/2004 - "To clarify the neuroprotective effects of melatonin as a free radical scavenger, we recorded changes in synaptic potentials and monitored the generation of superoxide (O)(2)(-) (using hydroethidine) in the CA1 pyramidal layers of rat hippocampal slices exposed to anoxia/aglycemia ('ischemic') stress. "
01/01/1995 - "Flow cytometric analysis of cell suspensions exposed to shock waves in the presence of the radical sensitive dye hydroethidine."
07/01/1994 - "After the application of shock waves, we have demonstrated cavitation-generated free radicals in cell-free solutions and also in the surviving and intact suspended MGH-U1 cells by hydroethidine measurements. "
01/01/1995 - "The fluorescence intensity of ethidium, the oxidised form of hydroethidine, was used for the flow-cytometric measurement of intracellular oxidising reagents present in RT4 tumour cells during shock-wave exposure. "
01/01/1995 - "The occurrence of intracellularly and extracellularly generated free radicals during shock wave exposure on an experimental Siemens lithotripter was tested with the radical sensitive dyes hydroethidine and dichlorofluorescin (DCFH). "
09/01/1986 - "A preliminary survey of various tumors indicated that uptake and accumulation of hydroethidine were dependent on concentration of the dye, duration of cell exposure to the dye, and metabolic state of the cells. "
07/01/2013 - "Furthermore, in the cell lines with the K-ras oncogene, overexpression of superoxide dismutases that detoxify non-mitochondrial sources of O 2.-, and treatment with the small molecule O 2.- scavenger Tempol, also decreased hydroethidine fluorescence, inhibited clonogenic survival and inhibited growth of tumor xenografts. "
|5.||Thiobarbituric Acid Reactive Substances
|7.||Amyloid (Amyloid Fibrils)
|8.||mitochondrial permeability transition pore
|10.||coenzyme Q10 (CoQ10)
|1.||Heterologous Transplantation (Xenotransplantation)