|1.||Richards, A Mark: 3 articles (12/2014 - 01/2008)|
|2.||Matsuzaki, Takashi: 2 articles (04/2014 - 10/2011)|
|3.||Lang, Florian: 2 articles (02/2014 - 04/2004)|
|4.||Gao, F: 2 articles (01/2014 - 04/2013)|
|5.||Zeng, K: 2 articles (01/2014 - 04/2013)|
|6.||Nakahari, Takashi: 2 articles (09/2012 - 09/2011)|
|7.||Iitaka, Daisuke: 2 articles (09/2012 - 09/2011)|
|8.||Okamoto, Kazuma: 2 articles (09/2012 - 09/2011)|
|9.||Komatsu, Shuhei: 2 articles (09/2012 - 09/2011)|
|10.||Marunaka, Yoshinori: 2 articles (09/2012 - 09/2011)|
|1.||Hypertension (High Blood Pressure)
01/01/2014 - "The aim of this study was to investigate the correlation between the natriuretic peptide precursor B (NPPB) gene single nucleotide polymorphism (SNP) c.-1298 G/T and pulse pressure (PP) of the Chinese Han population and the association between genotype and clinical indicators of hypertension. "
01/01/2014 - "In conclusion, there was no significant correlation between the NPPB gene c.-1298 G/T polymorphism and the incidence of essential hypertension in the Han population; however, the PP of the SNP c.-1298 GG genotype was greater than that of the GT+TT genotype in the control group."
01/01/2014 - "There were no significant differences in genotype frequency and distribution of the NPPB gene c.-1298 G/T polymorphism between the hypertension group and the control group (P>0.05); in the control group, the mean PP of individuals with the SNP c.-1298 GG genotype was greater than that of individuals with the GT+TT genotype (P<0.05). "
04/01/2013 - "A novel complex mutation in 5'-flanking sequence of the NPPB gene was detected in three patients (II 2, III 2, and III 5) of the hypertension family, which included c.-1195_ -1176 insert 5'-CCTTCTTTCTTTCTTTCTTT-3', c.-1208 T>A, c.-1214 T>C, and c.-1216 T>A. "
04/11/2000 - "No signs of systemic hypertension and ventricular hypertrophy are noted in Nppb(-/-) mice. "
|2.||Arteriovenous Malformations (Arteriovenous Malformation)
03/01/2004 - "The pathophysiological mechanisms and the salient features of normal perfusion pressure breakthrough (NPPB) are discussed on the basis of an operated case of arteriovenous malformation."
12/01/2001 - "A patient with normal perfusion pressure breakthrough (NPPB) after surgical removal of an arteriovenous malformation (AVM) was evaluated using single photon emission computed tomography cerebral blood flow (CBF) imaging. "
09/01/2000 - "To reduce the risk of surgical resection of giant arteriovenous malformation (AVM) and prevent normal perfusion pressure breakthrough (NPPB) and thus to lower postoperative mortality. "
03/01/1999 - "To reduce the risk of surgical resection of giant arteriovenous malformation (AVM) (> 6.0 cm) and prevent normal perfusion pressure breakthrough (NPPB) for lowering the postoperative mortality. "
10/01/1996 - "Obliteration procedures for large high-flow arteriovenous malformations (AVM) were simulated using a compartmental flow model to investigate the role of altered autoregulatory conditions in the development of hyperperfusion and normal perfusion pressure breakthrough (NPPB). "
06/28/2010 - "Immediately following surgical excision of a cerebral AVM, even normal brain tissue surrounding the lesion is subject to hemorrhage, a phenomenon termed normal perfusion pressure breakthrough (NPPB) syndrome. "
06/01/1998 - "Control of hemorrhage during AVM surgery is one of the key issues to prevent NPPB. "
11/01/1999 - "The 'normal perfusion pressure breakthrough' (NPPB) theory states that chronic hypoperfusion around AVMs induces the loss of autoregulatory capability; following AVM shunt obliteration, perfusion pressure elevation induces an increase in flow, due to 'vasomotor paralysis', which can cause hemorrhage. "
07/01/2015 - "The normal perfusion pressure breakthrough (NPPB) theory described by Spetzler in 1978 was adopted to explain the edema and hemorrhage that sometimes occur after resection of brain arteriovenous malformations (AVMs). "
01/01/2012 - "Two hypotheses, normal perfusion pressure breakthrough (NPPB) and occlusive hyperemia, prevail in the literature regarding the occasional development of hemorrhage and edema following AVM resection. "
03/19/2002 - "Pharmacological data suggest that both sorbitol and taurine are transported via a single malaria-induced pathway, which is sensitive to inhibition by 5-nitro-2-(3-phenylpropylamino)benzoic acid (IC(50) approximately 7 microM). "
11/01/1995 - "In this study a series of 16 arylaminobenzoates, analogues of the Cl- channel blocker 5-nitro-2-(3-phenylpropylamino)benzoic acid (NPPB), were tested for their effects on the transport of choline, a univalent cation, into malaria-infected cells. "
02/04/1994 - "For all of the substrates tested the order of potency of these three inhibitors was the same (NPPB > furosemide > niflumate) and dose-response curves for the effect of these inhibitors on malaria-induced choline transport were similar to those for malaria-induced thymidine transport. "
02/04/1994 - "5-Nitro-2-(3-phenyl-propylamino)benzoic acid (NPPB), furosemide, and niflumate blocked the malaria-induced transport of monovalent cations, neutral amino acids, sugars, nucleosides, and monovalent anions. "
|5.||Intracranial Arteriovenous Malformations (Cerebral Arteriovenous Malformation)
10/01/1996 - "[Hemodynamic simulation study of cerebral arteriovenous malformations: changes of wall stress and early detection of NPPB]."
01/01/1997 - "The syndrome of normal pressure perfusion breakthrough (NPPB) follows the surgical resection of a small fraction of cerebral arteriovenous malformations (AVM). "
07/26/2004 - "Restoration of normal perfusion pressure after resection of cerebral arteriovenous malformations (AVMs) is sometimes complicated by unexplained postoperative brain swelling and/or intracranial hemorrhage, which has been termed normal perfusion pressure breakthrough (NPPB). "
|3.||Chloride Channels (Chloride Channel)
|4.||Choline (Choline Chloride)
|6.||4,4'- Diisothiocyanostilbene- 2,2'- Disulfonic Acid (DIDS)
|8.||Brain Natriuretic Peptide (Natrecor)
|10.||Benzoic Acid (Ucephan)