|1.||Lamoureux, Francois: 2 articles (07/2013 - 02/2013)|
|2.||Zoubeidi, Amina: 2 articles (07/2013 - 02/2013)|
|3.||Kempf, Christoph: 2 articles (09/2012 - 12/2002)|
|4.||Ros, Carlos: 2 articles (09/2012 - 12/2002)|
|5.||Nagatomi, Ryoichi: 2 articles (04/2010 - 08/2009)|
|6.||Hatachi, Yukimasa: 2 articles (04/2010 - 08/2009)|
|7.||Kubo, Hiroshi: 2 articles (04/2010 - 08/2009)|
|8.||Yasuda, Hiroyasu: 2 articles (04/2010 - 08/2009)|
|9.||Nishimura, Hidekazu: 2 articles (04/2010 - 08/2009)|
|10.||Asada, Masanori: 2 articles (04/2010 - 08/2009)|
|1.||Colonic Neoplasms (Colon Cancer)
05/01/2009 - "To conclude, inhibition of macroautophagy by bafilomycin A(1) lowers G(1)-S transition and induces apoptosis in colon cancer cells. "
05/01/2009 - "In the present study, bafilomycin A(1), a vacuolar type H(+)-ATPase inhibitor, suppresses macroautophagy by preventing acidification of lysosomes in colon cancer cells. "
05/01/2001 - "We examined mucin sulfation and expression of oncofetal Thomsen-Friedenreich (TF) antigen (galactosyl beta1-3N-acetylgalactosamine alpha-) in the goblet cell-differentiated human colon cancer cell line LS174T following treatment with bafilomycin A(1, )which raises intra-Golgi pH, or monensin, which disrupts medial-trans Golgi transport. "
09/01/2012 - "Bafilomycin A(1) (BafA1) and NH(4)Cl, which raise endosomal pH, blocked the infection by preventing endosomal escape, resulting in a massive accumulation of capsids in the lysosomes. "
02/01/2011 - "Bafilomycin A(1) had no significant effect on the outcome of infection in insect cells."
02/01/2011 - "Bafilomycin A(1) was found to block the expression of a virus-encoded reporter gene in both infection and transfection of BHK cells. "
02/01/2011 - "Bafilomycin A(1) has been commonly used as a method of inhibition of infection by viruses known or suspected to follow the path of receptor-mediated endocytosis and low-pH-mediated membrane fusion. "
07/01/2003 - "Infection was also inhibited by blocking the acidification of endosomes by NH(4)Cl and bafilomycin A. "
12/01/2014 - "10 ng/mL TGF-β1 reduced the apoptosis incidence in the cells under serum starvation for 48 h, down-regulated the autophagy incidence in the cells pretreated with 3-methyladenine (3-MA) or Bafilomycin A (Baf A), partly rescued the increased apoptosis incidence in the cells pretreated with 3-MA, while further reduced the decreased apoptosis incidence in the cells pretreated with Baf A. "
02/01/2010 - "Indeed, a high percentage of the accumulated autophagic vesicles contain ubiquitin-positive contents, a feature that is not observed in autophagic vesicles that accumulate following starvation or treatment with Bafilomycin A. "
10/01/2009 - "Cardiomyocytes from starved mice treated with bafilomycin A(1) showed marked accumulation of lysosomes, and the myocardial amino acid content, which increased during starvation in normally fed mice, as well as the myocardial ATP content, were severely reduced, which likely contributed to the cardiac dysfunction. "
02/15/2013 - "Blocking autophagy using pharmacologic inhibitors (3-methyladenine, chloroquine, and bafilomycin A) or genetic inhibitors (siRNA targeting Atg3 and Atg7) enhanced cell death induced by Akt inhibitor AZD5363 in these tumor prostate cell lines. "
01/01/2000 - "Bafilomycin A(1), a selective inhibitor of V-type H(+)-translocating ATPase (V-ATPase), may be a useful adjunct in cancer chemotherapy (Altan et al.  J Exp Med 187:1583-1598). "
|1.||Proton-Translocating ATPases (ATPase, H+)
|3.||Adenosine Triphosphatases (ATPase)
|6.||Small Interfering RNA (siRNA)
|7.||Vacuolar Proton-Translocating ATPases (V-Type ATPase)
|1.||Drug Therapy (Chemotherapy)