|1.||Sharova, T S: 1 article (05/2015)|
|2.||Shpakov, A O: 1 article (05/2015)|
|3.||Pertseva, M N: 1 article (05/2015)|
|4.||Kuznetsova, L A: 1 article (05/2015)|
|5.||Shalaby, A: 1 article (03/2015)|
|6.||Malek, Hala Abdel: 1 article (03/2015)|
|7.||Jiang, Zuo-Feng: 1 article (04/2014)|
|8.||Zhao, Qiang: 1 article (04/2014)|
|9.||Xu, Ding-Li: 1 article (04/2014)|
|10.||Li, Biao: 1 article (04/2014)|
08/01/1999 - "Maximal lipolytic responses to a specific beta(3)-AR agonist, BRL-37344, were significantly attenuated at post burn days (PBD) 3 and 7. Despite significant reduction of the cell surface beta(3)-AR number and its mRNA at PBD 3 and 7, BRL-37344 and forskolin-stimulated cAMP levels were not decreased. "
07/01/1999 - "The stress-induced fever was mimicked by central nervous system administration of the selective beta2-agonist, salbutamol, and the beta3-agonist, BRL 37344. "
07/01/1999 - "The purpose of this study was to determine the relative contribution of the different beta-receptor types to stress fever using beta1-, beta2-, and beta3-receptor subtype selective antagonists (atenolol [beta1], ICI-118551 [beta2], and SR 59230A [beta3]) and agonists (dobutamine [beta1], salbutamol [beta2], and BRL 37344 [beta3]) on the Tb of rats. "
04/01/2014 - "β3-Adrenoreceptor stimulation with BRL-37344 was associated with an increase in LA diameter and a decrease in LA function in chronic heart failure. "
04/01/2014 - "To investigate the effect of selective β3-adrenoreceptor agonist BRL-37344 on L-type Ca(2+) current (Ica,L) and mRNA expression of L-type Ca(2+) channel α2δ-2 (Cacna2d2) in rats with chronic heart failure (CHF). "
06/01/2005 - "To evaluate the effects of different doses of N(G)-nitro-L-arginine methyl ester (L-NAME) on hemodynamics, cyclic guanosine monophosphate (cGMP) production and the level of beta-adrenergic receptors (beta-ARs) mRNA in a heart failure rat model after BRL-37344 (beta(3)-ARs agonist) injection. "
02/01/1996 - "4. In vivo, the inhibition of colonic motility and the thermogenic response of brown adipose tissue elicited in rats by the selective beta 3-adrenoceptor agonists SR 58611A and BRL 37344 respectively were substantially diminished by the representative APAT, SR 59230A, at oral doses (< or = 5 mg kg-1) well below those half maximally effective (ID50) for preventing beta 1-(isoprenaline tachycardia > or = 80 mg kg-1) or beta 2-(salbutamol bronchodilatation, 44 mg kg-1) mediated responses. "
|1.||Messenger RNA (mRNA)
|4.||3- (2- ethylphenoxy)- 1- (1,2,3,4- tetrahydronaphth- 1- ylamino)- 2- propanol oxalate
|10.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)