|1.||de la Houssaye, Becky A: 1 article (01/2013)|
|2.||Friedman, Jacob E: 1 article (01/2013)|
|3.||Heerwagen, Margaret J R: 1 article (01/2013)|
|4.||Janssen, Rachel C: 1 article (01/2013)|
|5.||Stewart, Michael S: 1 article (01/2013)|
|6.||Kemp, Bruce E: 1 article (05/2008)|
|7.||Bruce, Clinton R: 1 article (05/2008)|
|8.||Macaulay, Lance: 1 article (05/2008)|
|9.||van Denderen, Bryce J W: 1 article (05/2008)|
|10.||Hoy, Andrew J: 1 article (05/2008)|
01/01/2013 - "E18.5 Fetuses from WT-HFD mothers had larger placentas (P<0.02), as well as increased placenta and fetal liver TG deposition (P<0.01 and P<0.02, respectively) and increased placental LPL TG-hydrolase activity (P<0.02), which correlated with degree of maternal insulin resistance (r = 0.59, P<0.02). "
05/01/2008 - "When ATGL protein expression and TG hydrolase activity in obese, insulin-resistant rats were restored to levels observed in lean rats, TG content was reduced; however, the insulin resistance induced by the high-fat diet persisted. "
05/01/2008 - "Although ATGL content and TG hydrolase activity are decreased in obese, insulin-resistant phenotypes, overexpression does not rescue the condition, indicating reduced ATGL is unlikely to be a primary cause of obesity-associated insulin resistance."