|1.||Bertozzi, Carolyn R: 5 articles (03/2012 - 09/2002)|
|2.||Leary, Julie A: 4 articles (03/2012 - 05/2003)|
|3.||Riley, Lee W: 3 articles (06/2011 - 09/2002)|
|4.||Mougous, Joseph D: 3 articles (08/2004 - 09/2002)|
|5.||Schelle, Michael W: 2 articles (03/2012 - 07/2007)|
|6.||Leavell, Michael D: 2 articles (07/2007 - 05/2003)|
|7.||Petzold, Christopher J: 2 articles (07/2007 - 08/2004)|
|8.||Lin, Fiona L: 2 articles (07/2007 - 08/2004)|
|9.||Cox, Jeffery S: 2 articles (07/2007 - 05/2003)|
|10.||Senaratne, Ryan H: 2 articles (08/2004 - 09/2002)|
03/09/2012 - "tuberculosis, suggesting an avenue for perturbing SL-1 biosynthesis in vivo. "
07/03/2007 - "tuberculosis confirmed their essential role in SL-1 biosynthesis and their order of action. "
08/01/2004 - "tuberculosis resulted in the loss of T2S and SL-1 formation, respectively. "
05/13/2003 - "tuberculosis, is required for SL-1 production. "
06/01/2011 - "tuberculosis, its cell wall may undergo changes in SL-1 and SL(1278) contents during a natural course of infection and this may serve as an important adaptive strategy for M. "
12/01/2011 - "We speculated that the effect of SL-1 on M.tb pathogenicity could be associated with long-term survival and persistence establishment during infection. "
09/01/2007 - "Microscopic observation revealed that infection of SL-1 cells with AfMNPV resulted in apoptosis, displaying apoptotic bodies in fluorescent-stained nuclei of AfMNPV-infected SL-1cells. "
07/03/2007 - "The loss of SL-1 (and SL(1278)) did not appear to affect bacterial replication or trafficking, suggesting that the functions of SL-1 are specific to human infection."
05/13/2003 - "Thus, we hypothesize that either MmpL8 transports molecules in addition to SL-1 that mediate host-pathogen interactions or the accumulation of SL(1278) in mmpL8 mutant cells interferes with other pathways required for growth during the early stages of infection."
05/13/2003 - "However, SL-1 per se is not required for establishing infection as pks2 mutants, which are defective in an earlier step in SL-1 biosynthesis, have no obvious growth defect. "
12/29/1997 - "These observations demonstrate that inappropriate expression of SL-1 initiates a cascade of events that may represent a coordinated program leading to loss of the differentiated epithelial phenotype and gain of some characteristics of tumor cells. "
09/01/1996 - "These observations suggest that SL-1 and other MMPs may be useful targets for therapeutic intervention in cancer."
|5.||Burkitt Lymphoma (Burkitt's Lymphoma)
|2.||Proteins (Proteins, Gene)
|5.||ascorbic acid 2-(11-oxoolean-12-en-29-oic acid ethyl ester 3-yl-phosphate)
|6.||adenylylsulfate kinase (Adenylyl-sulfate kinase)