|1.||Li, Yue: 1 article (07/2014)|
|2.||Ye, Yong: 1 article (07/2014)|
|3.||Fang, Fei: 1 article (07/2014)|
|4.||Edling, Charlotte E: 1 article (05/2014)|
|5.||Falasca, Marco: 1 article (05/2014)|
|6.||Maffucci, Tania: 1 article (05/2014)|
|7.||Selvaggi, Federico: 1 article (05/2014)|
|8.||Ghonaim, Ragheda: 1 article (05/2014)|
|9.||Canini, Frédéric: 1 article (03/2006)|
|10.||Bourdon, Lionel: 1 article (03/2006)|
01/01/1991 - "Influence of CGS 15943 A (a nonxanthine adenosine antagonist) on the protection offered by a variety of antiepileptic drugs against maximal electroshock-induced seizures in mice."
01/01/1991 - "CGS 15943 A (a nonxanthine adenosine antagonist) was studied on the protective efficacy of carbamazepine (60 min prior to the convulsive test), diazepam (60 min), diphenylhydantoin (120 min), phenobarbital (120 min), and valproate (30 min) against maximal electroshock-induced convulsions in mice. "
07/02/2014 - "The results showed that the sapogenin and its derivative increased dopamine content in striatum and tyrosine hydroxylase (TH) positive cells in substantia nigra and relieved inflammation and behavioral disorder, but the effect on movement was reversed by dopamine receptor antagonist haloperidol and was not intervened by adenosine receptor antagonist CGS 15943. "
03/01/2006 - "Thus it might be suggested that the suramin/CGS-15943 blunting of hypoxia-induced hypothermia would be specific to hypoxia-induced mechanisms."
03/01/2006 - "Suramin/CGS-15943 treatment blunted the profound hypothermia observed in control rats throughout the hypoxia exposure, whereas CGS-15943 treatment blunted hypothermia during only 3 h, and suramin treatment had no effect. "
03/01/2006 - "In normoxia, suramin/CGS-15943 induced an increase in T(CO) but to a far lesser extent than observed in hypoxia. "
01/01/1995 - "Respiratory frequency (f) and minute volume (VE) increased during 15-minute periods of hypoxia, and intramuscular administration of caffeine (0.3 and 1.0 mg/kg), paraxanthine (0.3 and 1.0 mg/kg) and CGS 15943 (0.03 and 0.1 mg/kg) attenuated the ventilatory response to hypoxia. "
01/01/1995 - "Doses of caffeine, paraxanthine or CGS 15943 that attenuated the ventilatory response to hypoxia had no significant effect on f or VE during conditions of normoxia or hypercapnia. "
01/01/1994 - "CGS 15943 markedly reduced stroke injury assessed by locomotor activity monitoring and by histopathological measurement of hippocampal CA1 pyramidal cell injury. "
01/01/1994 - "The adenosine A2 receptor antagonist CGS 15943 (0.1 mg/kg, i.p.) was tested for cerebroprotective activity in a gerbil stroke model. "
|2.||Purinergic P1 Receptors (Adenosine Receptor)
|3.||Valproic Acid (Valproate, Semisodium)
|4.||Tyrosine 3-Monooxygenase (Tyrosine Hydroxylase)
|5.||Dopamine Receptors (Dopamine Receptor)