|1.||Blessing, Karen: 3 articles (06/2011 - 05/2010)|
|2.||Kador, Peter F: 3 articles (06/2011 - 05/2010)|
|3.||Reinhardt, Richard A: 2 articles (06/2011 - 05/2010)|
|4.||O'Meara, James D: 1 article (06/2011)|
|5.||Marx, David B: 1 article (06/2011)|
|6.||Makita, Jun: 1 article (01/2011)|
|7.||Randazzo, James: 1 article (01/2011)|
|8.||Zhang, Peng: 1 article (01/2011)|
|9.||Hamada, Tomofumi: 1 article (05/2010)|
01/01/1998 - "AL1576 showed only partial protection against the cataract induced by 500 microM ND. "
01/01/2011 - "As anticipated, treatment with AL1576 prevented cataract by inhibiting sorbitol formation in the lens. "
08/01/1998 - "Cataract formation was inhibited in both diabetic and galactosemic rats by either Ponalrestat or AL 1576. "
01/01/1992 - "While the molecular mechanism of this cataract is unclear, it has recently been demonstrated that the aldose reductase inhibitor ALO1576 can prevent lens opacification in this system. "
06/01/1988 - "Topical treatment with the aldose reductase inhibitor, AL 1576, begun immediately after diabetes induction, prevents endothelial cell changes and cataract formation. "
05/01/2010 - "In studies conducted in young normal control and streptozotocin diabetic rats (100 g) treated with and without the aldose reductase inhibitor (ARI) imirestat, experimental periodontitis was induced in one side of the mouth by 3 injections of lipopolysaccharide (LPS) from Escherichia coli 055:B5 9 into the palatal gingiva between the first and second maxillary molars at 48-hour intervals. "
06/01/2011 - "Periodontitis was induced with three consecutive palatal injections of Porphyromonas gingivalis lipopolysaccharide (LPS) at 48-hour intervals between the first and second molars on the right side in young, age-matched, streptozotocin-induced rats with and without diabetes 44 days after initiation of diets with and without the ARIs tolrestat, imirestat, and quercetin. "
08/01/1997 - "The continuous presence of AL-1576, an aldose reductase inhibitor (ARI), normalized the effect of severe hyperglycemia on Rb+ uptake in the chronic (28 days) but not the acute exposure protocols. "
01/01/1997 - "Following the demonstration of increased prostaglandin E (PGE) levels in glomeruli from diabetic rats (14.9 +/- 2.5 v 59.1 +/- 19.4 ng PGE/mg protein), a specific inhibitor of aldose reductase, HOE-843, was used in vitro to analyze the response to hyperglycemia of the steps preceding prostaglandin production. "
|4.||Aldehyde Reductase (Aldose Reductase)
|9.||Staphylococcal Protein A (A, Protein)