|1.||Michetti, Fabrizio: 6 articles (01/2015 - 03/2004)|
|2.||Geloso, Maria Concetta: 5 articles (01/2015 - 03/2004)|
|3.||Corvino, Valentina: 5 articles (01/2015 - 03/2004)|
|4.||Kato, N: 4 articles (03/2005 - 06/2001)|
|5.||Marchese, Elisa: 3 articles (01/2015 - 07/2012)|
|6.||Lattanzi, Wanda: 3 articles (01/2015 - 07/2012)|
|7.||Giannetti, Stefano: 3 articles (01/2014 - 02/2010)|
|8.||Shim, Hyun Soo: 3 articles (04/2012 - 09/2011)|
|9.||Shim, Insop: 3 articles (04/2012 - 09/2011)|
|10.||Park, Hyun-Jung: 3 articles (04/2012 - 09/2011)|
|1.||Memory Disorders (Memory Loss)
04/01/2012 - "The present study examined the effects of Tremella fuciformis (TF) on the learning and memory function and the neural activity in rats with trimethyltin (TMT)-induced memory deficits. "
03/01/2012 - "The present study examined the effects of krill-derived phosphatidylserine (Krill-PS) on the learning and memory function and the neural activity in rats with trimethyltin (TMT)-induced memory deficits. "
09/01/2011 - "Neuroprotective Effect of Lucium chinense Fruit on Trimethyltin-Induced Learning and Memory Deficits in the Rats."
09/01/2008 - "Peroral administration of a single dose of trimethyltin (8.5 mg/kg) induced spatial memory loss and the extensive loss of CA3 pyramidal neurons in hippocampi, as indicated by the results of a Morris water maze task and histologic examination, respectively. "
12/01/2012 - "Trimethyltin (TMT) is an organotin neurotoxicant with effects that are selectively localized to the limbic system (especially the hippocampus), which produces memory deficits and temporal lobe seizures. "
01/01/1991 - "Experiments where the slice has been used to study the neurotoxin trimethyltin are discussed, along with its use as a model for investigating the neurodegenerative problems associated with hypoxia, ischemia, and epilepsy."
05/01/1999 - "2. In this study, we characterized the pharmacological effect mediated by sigma ligands on two lesional models of amnesia in mice: (1) the hypoxia-related learning and memory impairment model induced by repeated exposure to carbon monoxide (CO) gas; and (2) the intoxication with trimethyltin (1 mg kg(-1)). "
05/01/2007 - "We extend our earlier findings by showing that DM attenuated trimethyltin (TMT)-induced neurotoxicity (convulsions, hippocampal degeneration and spatial memory impairment) in rats. "
06/29/2001 - "Trimethyltin (TMT), an organic metal, has been known to induce behavioral abnormalities including seizures and aggression. "
12/01/1997 - "Trimethyltin may offer potential as a tool for investigations on the relationship between neuronal death in the hippocampus and the development of seizure susceptibility and learning impairment. "
12/01/1997 - "The maximal seizure susceptibility at four days post-trimethyltin was confirmed by the immediate and long-lasting appearance of spike discharge in the hippocampus. "
12/01/1997 - "Trimethyltin syndrome as a hippocampal degeneration model: temporal changes and neurochemical features of seizure susceptibility and learning impairment."
09/15/1999 - "An acute administration of the hippocampal toxicant trimethyltin (TMT) produced a specific pattern of neuronal necrosis in dentate granule cells with accompanying astrogliosis and initiation of a cytokine response within 24 hours. "
11/01/1996 - "Trimethyltin (TMT) is a toxic alkyltin that produces neuronal necrosis in the CNS. "
07/01/1995 - "Trimethyltin (TMT) is a toxic alkyltin compound that is known to produce neuronal necrosis in the CNS. "
10/01/1998 - "To study the role of cytokines in the early stages of brain injury, we examined alterations in the 17-day-old mouse hippocampus during trimethyltin-induced neurodegeneration characterized by neuronal necrosis, microglia activation in the dentate, and astrocyte reactivity throughout the hippocampus. "
01/01/1988 - "The main results of toxicity are skin and eye irritation; cholangitis of the lower biliary tract, and later hepatotoxicity; and neurotoxicity, which has been shown to be due to intramyelin oedema induced by triethyltin, and neuronal necrosis caused by trimethyltin. "
|5.||Neurodegenerative Diseases (Neurodegenerative Disease)
06/01/2008 - "Trimethyltin (TMT) intoxication is considered a suitable experimental model to study the molecular basis of selective hippocampal neurodegeneration as that occurring in several neurodegenerative diseases. "
12/01/2011 - "Trimethyltin (TMT) is an organotin compound with neurotoxic effects on the hippocampus and other limbic structures and is used to model neurodegenerative diseases targeting these brain areas. "
02/15/2010 - "Trimethyltin (TMT) is an organotin compound that generates neurotoxic effects, and it has been used in a model of neurodegenerative disease and memory dysfunction. "
10/15/1997 - "Endogenous excitatory amino acids (EAAs) such as glutamic or aspartic acids have been proposed to mediate the brain damage to EAA receptor-rich brain sites that is caused by a variety of external toxic agents (glutamic acid, domoic acid, kainic acid, ibogaine, trimethyltin (TMT), 3-nitropropionic acid (3-NPA)), as well as from such naturally-occurring age-related neurodegenerative diseases as Alzheimer's disease, Huntington's chorea, and Parkinson's disease. "
|1.||Kainic Acid (Kainate)
|3.||Photosensitizing Agents (Photosensitizers)
|5.||NF-kappa B (NF-kB)
|8.||L-Lactate Dehydrogenase (Lactate Dehydrogenase)