|1.||Rajamani, Rathinam: 2 articles (05/2006 - 02/2006)|
|2.||Muthuvel, Arumugham: 2 articles (05/2006 - 02/2006)|
|3.||Sheeladevi, Rathinasamy: 2 articles (05/2006 - 02/2006)|
|4.||Manikandan, Sundaramahalingam: 1 article (05/2006)|
|5.||Braun, U: 1 article (02/2005)|
|6.||Bleul, U: 1 article (02/2005)|
|7.||Bachofner, C: 1 article (02/2005)|
|8.||Stocker, H: 1 article (02/2005)|
|9.||Hässig, M: 1 article (02/2005)|
|10.||Bouldin, Thomas W: 1 article (04/2002)|
08/01/1996 - "Carbicarb is a new alkalinizing agent that has been proposed as a replacement for NaHCO3 in the treatment of metabolic acidosis. "
02/01/1996 - "The data demonstrate that 1) both Carbicarb and NaHCO3 significantly increase arterial pCO2; and 2) use of either alkalinizing agent in moderate acidosis does not alter the course of acidosis."
10/01/1994 - "However, the potential therapeutic advantage of Carbicarb remains to be determined, especially in patients with more severe metabolic acidosis."
10/01/1994 - "Because of these characteristics, Carbicarb may be a more suitable agent than bicarbonate in the treatment of metabolic acidosis. "
05/01/1989 - "These data demonstrate that bicarbonate therapy of systemic acidosis may be associated with "paradoxical" intracellular brain acidosis, whereas Carbicarb causes both systemic and intracellular alkalinization under conditions of fixed ventilation."
02/01/1996 - "The present study was designed to investigate whether Carbicarb or NaHCO3 is superior to normal saline in the treatment of hypoxic lactic acidosis. "
12/01/1989 - "Brain pH effects of NaHCO3 and Carbicarb in lactic acidosis."
01/01/1988 - "We studied the effects of carbicarb in an animal preparation of hypoxic lactic acidosis (HLA). "
02/01/1996 - "Effects of Carbicarb and sodium bicarbonate on hypoxic lactic acidosis in newborn pigs."
09/01/1993 - "The data suggest that Carbicarb administration in hypoxic lactic acidosis improved hemodynamics compared with sodium bicarbonate or sodium chloride administration. "
01/01/1988 - "The liver pHi (normal = 6.99, hypoxia = 6.80) improved with carbicarb (to 6.92), but decreased further with NaHCO3 (to 6.40). "
02/01/1996 - "When the pH fell to < 7.2, hypoxia was reversed by placing the animals in 21% O2 (experiment 1) or 100% O(2) (experiment 2) and either saline, Carbicarb or NaHCO3 were given. "
01/01/1988 - "Improved hemodynamic function during hypoxia with Carbicarb, a new agent for the management of acidosis."
01/01/1988 - "Dogs with HLA (n = 28) were then treated with 2.5 meq/kg of either NaHCO3 or carbicarb over 1 hr. Measurements were made, after 1 hr of hypoxia and 1 hr of therapy, of: cardiac hemodynamics, blood gases, liver intracellular pH (pHi), oxygen consumption, and regional lactate production. "
04/01/1991 - "Metabolic acidosis (pH 7.20) and alkalosis (pH 7.60) were induced with HCl and Carbicarb (isosmolar Na2CO3 and NaHCO3), respectively. "
01/01/2001 - "Our results suggest that agents such as sodium bicarbonate, Carbicarb and the diuretic furosemide--which are known to induce metabolic alkalosis in humans--may be useful in enhancing the efficacy of these treatment regimens in humans."
|5.||Heart Arrest (Cardiac Arrest)
04/01/2002 - "Low-dose Carbicarb improves cerebral outcome after asphyxial cardiac arrest in rats."
04/01/2002 - "Rats were prospectively randomized to either a control (n=45), low-dose Carbicarb (LDC; 3 mL/kg, n=45), or high-dose Carbicarb (HDC; 6 mL/kg, n=45) group in a blinded fashion during resuscitation after 8 minutes of asphyxial cardiac arrest. "
03/01/1991 - "In this model of cardiac arrest, carbicarb was not superior to sodium bicarbonate in the correction of metabolic acidosis during CPR."
04/01/2002 - "The objective of this study was to determine whether attenuation or neutralization of cerebral acidosis by Carbicarb alters hippocampal glutamate levels, neuronal cell death, and neurologic deficits after reperfusion from asphyxial cardiac arrest in rats. "
12/01/1996 - "The topics discussed in this session include a partial review of laboratory and clinical studies examining the effects of adrenergic agonists on restoration of spontaneous circulation after cardiac arrest, the effects of varying doses of epinephrine, and the effects of novel vasopressors, buffer agents (NaHCO3, THAM, 'Carbicarb') and anti-arrhythmics (lidocaine, bretylium, amiodarone) in refractory ventricular fibrillation. "
|1.||Sodium Bicarbonate (Baking Soda)
|2.||Sodium Chloride (Saline Solution)
|5.||Glutamic Acid (Glutamate)
|10.||Adrenergic Agonists (Adrenergic Receptor Agonist)
|2.||Cardiopulmonary Resuscitation (CPR)