|1.||Fatemi, S Hossein: 1 article (08/2014)|
|2.||Folsom, Timothy D: 1 article (08/2014)|
|3.||Reutiman, Teri J: 1 article (08/2014)|
|4.||Rooney, Robert J: 1 article (08/2014)|
|5.||Thuras, Paul D: 1 article (08/2014)|
|6.||Rustan, Oyvind G: 1 article (08/2014)|
|7.||Bergvall, K: 1 article (01/2012)|
|8.||Wilhelm, S: 1 article (01/2012)|
|9.||Adriaensen, Dirk: 1 article (01/2012)|
|10.||Beco, L: 1 article (01/2012)|
04/01/1997 - "The present study investigated the involvement of N-methyl-D-aspartate (NMDA) receptor in the prostaglandin-induced hyperalgesia by use of mice tacking NMDA receptor epsilon 1, epsilon 4, or epsilon 1/epsilon 4 subunits. "
03/23/2001 - "PGE(2)- and NMDA-induced allodynia were observed in NMDA receptor epsilon 4 (NR2D) subunit knockout (GluR epsilon 4(-/-)) mice, but not in epsilon 1 (NR2A) subunit knockout (GluR epsilon 1(-/-)) mice. "
04/01/1997 - "Absence of prostaglandin E2-induced hyperalgesia in NMDA receptor epsilon subunit knockout mice."
07/01/2003 - "Spinal intrathecal administration of PGF2alpha to mouse induces touch-evoked pain (mechanical allodynia), in which capsaicin-insensitive primary afferent Abeta-fibres and N-methyl-d-aspartate receptor epsilon 4 subunit are involved. "
03/23/2001 - "The induction of allodynia by PGE(2) and NMDA was abolished by the NMDA receptor epsilon 2 (NR2B) antagonist CP-101,606 and neonatal capsaicin treatment. "
|2.||Congenital Myasthenic Syndromes (Myasthenia Gravis, Congenital)
01/01/2004 - "[Congenital myasthenic syndrome related to homozygous missense mutation in promoter region of acetylcholine receptor epsilon-subunit gene]."
12/01/2003 - "A frameshifting 7 bp deletion (epsilon553del7) in exon 7 of CHRNE encoding the acetylcholine receptor epsilon subunit, observed in seven congenital myasthenic syndrome patients, enhances expression of an aberrantly spliced transcript that skips the preceding 101 bp exon 6. To recapitulate the aberrant splicing, we cloned the entire CHRNE spanning 12 exons and 11 introns and expressed it in COS cells. "
11/01/2002 - "Congenital myasthenic syndrome due to heteroallelic nonsense/missense mutations in the acetylcholine receptor epsilon subunit gene."
05/01/2002 - "Congenital myasthenic syndromes (CMSs) are frequently caused by mutations of the coding region of the acetylcholine receptor epsilon subunit (AChRepsilon) gene leading to a reduced expression of the acetylcholine receptor (AChR) at the postsynaptic membrane. "
05/01/1999 - "Congenital myasthenic syndrome caused by a mutation in the Ets-binding site of the promoter region of the acetylcholine receptor epsilon subunit gene."
02/01/2002 - "A novel lipid raft-associated glycoprotein, TEC-21, activates rat basophilic leukemia cells independently of the type 1 Fc epsilon receptor."
03/10/1994 - "Effects of herbimycin A and ST638 on Fc epsilon receptor-mediated histamine release and Ca2+ signals in rat basophilic leukemia (RBL-2H3) cells."
02/01/1992 - "Binding of monoclonal antibody AA4 to gangliosides on rat basophilic leukemia cells produces changes similar to those seen with Fc epsilon receptor activation."
11/01/1989 - "This IgE is both a solution receptor and, when anchored to its high affinity Fc epsilon receptor on rat basophilic leukemia (RBL) cells, a surface receptor. "
09/20/1988 - "These bilayers were reconstituted with purified IgE-Fc epsilon receptor complex and the intact 110-kDa channel-forming protein, both isolated from plasma membranes of rat basophilic leukemia cells (line RBL-2H3). "
05/01/1991 - "Significance of soluble Fc epsilon receptor II (sFc epsilon RII/CD23) in serum and possible application of sFc epsilon RII for the prevention of allergic reactions."
01/01/1990 - "Fc epsilon receptor II-positive macrophages and platelets: potent effector cells in allergy and defence against helminth parasites."
05/14/2010 - "The high affinity receptor for IgE, Fc epsilon receptor I (FcepsilonRI), is an activating immune receptor and key regulator of allergy. "
12/01/1993 - "Allergy-associated I epsilon and Ec epsilon receptor II (CD23b) genes activated via binding of an interleukin-4-induced transcription factor to a novel responsive element."
08/01/1992 - "[IgE-mediated allergy and Fc epsilon receptor II]."
|5.||Atopic Dermatitis (Atopic Eczema)
01/01/1992 - "Fc epsilon receptor II/CD23+ lymphocytes in atopic dermatitis. "
05/01/1991 - "Fc epsilon receptor II/CD23-positive lymphocytes in atopic dermatitis. "
11/01/1990 - "Fc epsilon receptor II/CD23 positive lymphocytes in atopic dermatitis: II. Infiltration of Fc epsilon R II(+) T cells in the skin lesion."
03/01/1990 - "[Serum IgE and Fc epsilon receptor positive lymphocytes in atopic dermatitis--relation to the background of respiratory atopy]."
01/01/1997 - "Therefore, the conclusion can be drawn that, in patients with atopic dermatitis, there are no protein sequence differences within the Fc part of the IgE which could be responsible for distinct functional features with regard to Fc epsilon-receptor binding and signal transduction or could account for the frequent occurrence of anti-IgE autoantibodies in these individuals."
|2.||Immunoglobulin E (IgE)
|3.||Acetylcholine (Acetylcholine Chloride)
|4.||IgE Receptors (Fc epsilon RI)
|5.||Interleukin-4 (Interleukin 4)
|6.||Messenger RNA (mRNA)
|7.||Morphine (MS Contin)
|8.||Cromolyn Sodium (Cromoglicic Acid)
|10.||beta-Endorphin (Endorphin, beta)