|1.||Cook, J M: 2 articles (08/2001 - 04/2001)|
|2.||Cook, James: 1 article (11/2002)|
|3.||Ma, Chunrong: 1 article (11/2002)|
|4.||Rowlett, James K: 1 article (11/2002)|
|5.||Spealman, Roger D: 1 article (11/2002)|
|6.||Platt, Donna M: 1 article (11/2002)|
|7.||Bergman, J: 1 article (08/2001)|
|8.||Cox, E D: 1 article (08/2001)|
|9.||Paronis, C A: 1 article (08/2001)|
|10.||Rowlett, J K: 1 article (04/2001)|
08/01/2001 - "Under the schedule of stimulus shock-termination, both flumazenil and beta-CCt antagonized zolpidem and zaleplon; however, the effects of beta-CCt were less consistent than the effects of flumazenil. "
09/01/1999 - "Under the schedule of stimulus-shock termination, midazolam produced dose-related decreases in response rate, and these effects were surmountably antagonized by flumazenil, bretazenil, Ro 41-7812, Ro 42-8773, and beta-CCt. "
11/01/2002 - "Flumazenil antagonized all of the behavioral effects of zolpidem and triazolam, whereas beta-CCt antagonized only zolpidem- and triazolam-induced ataxia. "
04/01/2001 - "The ability of zolpidem, but not triazolam or diazepam, to suppress USVs and augment hypothermia was antagonized by beta-CCt, whereas the increase in motor incoordination engendered by zolpidem, triazolam, and diazepam was not sensitive to beta-CCt administration. "
09/01/1999 - "beta-CCT which was devoid of activity per se, antagonized the effects of the non-selective BZ (omega) receptor full agonist diazepam and the selective BZ(1) (omega(1)) receptor full agonist zolpidem against seizures produced by isoniazid, but beta-CCT failed to affect their action on seizures produced by pentylenetetrazole (PTZ), suggesting that BZ(2) (omega(2)) receptors may be primarily involved in the convulsant action of PTZ. "