|1.||Galvis, Alvaro E: 1 article (06/2014)|
|2.||Fan, Hung: 1 article (06/2014)|
|3.||Fisher, Hugh E: 1 article (06/2014)|
|4.||Camerini, David: 1 article (06/2014)|
|5.||Nitta, Takayuki: 1 article (06/2014)|
|6.||Mayanja-Kizza, H: 1 article (11/2005)|
|7.||Baseke, J: 1 article (11/2005)|
|8.||Abraha, A: 1 article (11/2005)|
|9.||Peters, P: 1 article (11/2005)|
|10.||Aung, H: 1 article (11/2005)|
06/01/2014 - "Moreover, minus-strand strong-stop DNA rapidly accumulated in the cytoplasm in the first 2 h of infection but shifted to the nuclear fraction by 6 h postinfection. "
11/01/2005 - "HIV-1 strong stop DNA was assessed by real-time polymerase chain reaction (PCR) as a measure of infection. "
11/01/2004 - "Viral exposure to RMAD-4 for 1 h prior to infection reduced HIV minus-strand strong stop DNA and HIV cDNA by 4- to 20-fold during the first round of replication, showing that RMAD-4-exposed virions were not entering cells during the first 24 h. "
07/01/2002 - "PCR analysis of newly made viral DNA upon infection with virions containing any of the three aptamers with the strongest binding affinities (70.8,13, 70.15, and 80.55,65) showed that all three were able to form the minus-strand strong-stop DNA. "
03/01/2001 - "HIV-1 infection of CAEC was abortive with minimal reverse transcription of strong stop DNA and pol but not full-length or two LTR DNA circles. "
|1.||Complementary DNA (cDNA)
|3.||DNA (Deoxyribonucleic Acid)