|1.||Dahl, Niklas: 8 articles (01/2011 - 05/2003)|
|2.||Flygare, Johan: 6 articles (12/2014 - 05/2003)|
|3.||Karlsson, Stefan: 6 articles (12/2014 - 05/2003)|
|4.||Badhai, Jitendra: 6 articles (01/2011 - 01/2009)|
|5.||Schuster, Jens: 6 articles (01/2011 - 01/2009)|
|6.||Richter, Johan: 5 articles (12/2011 - 05/2003)|
|7.||Fröjmark, Anne-Sophie: 5 articles (06/2010 - 01/2009)|
|8.||Tchernia, Gil: 5 articles (11/2008 - 01/2003)|
|9.||Da Costa, Lydie: 5 articles (11/2008 - 01/2003)|
|10.||Dianzani, Irma: 4 articles (10/2014 - 12/2004)|
|1.||Kidney Diseases (Kidney Disease)
|2.||Diamond-Blackfan Anemia (Anemia, Diamond Blackfan)
10/01/2014 - "Diamond-Blackfan anemia, characterized by defective erythroid progenitor maturation, is caused in one-fourth of cases by mutations of ribosomal protein S19 (RPS19), which is a component of the ribosomal 40S subunit. "
01/01/2014 - "A Case of Diamond Blackfan Anemia (DBA) with Mutation in Ribosomal Protein S19."
12/01/2011 - "Mice with ribosomal protein S19 deficiency develop bone marrow failure and symptoms like patients with Diamond-Blackfan anemia."
06/15/2010 - "Heterozygous mutations in the ribosomal protein S19 (RPS19) gene are associated with Diamond-Blackfan anemia (DBA). "
02/01/2010 - "Defects in the RPS19 gene, encoding the ribosomal protein S19, are the main known cause of Diamond-Blackfan anemia and account for more than 25% of cases. "
01/01/2011 - "We next examined the involvement of the cross-linked ribosomal protein S19 dimer (RP S19), which is released during apoptosis, in the acquisition of tumor immunity. "
01/01/2011 - "Roles of the ribosomal protein S19 dimer and chemically induced apoptotic cells as a tumor vaccine in syngeneic mouse transplantation models."
01/01/2012 - "Regions statistically significantly associated with CIN3/cancer included genes for peroxiredoxin 3 PRDX3, and ribosomal protein S19 RPS19. "
02/15/1992 - "Three complementary DNA encoding S19 ribosomal protein (S19), laminin-binding protein (LBP), and HLA class I (HLA-I) genes were isolated from a colon tumor-enriched subtraction library. "
12/01/2013 - "Involvement of regional neutrophil apoptosis promotion by ribosomal protein S19 oligomers in resolution of experimental acute inflammation."
11/01/2000 - "Molecular mechanism of monocyte predominant infiltration in chronic inflammation: mediation by a novel monocyte chemotactic factor, S19 ribosomal protein dimer."
02/15/1992 - "Differential expression of S19 ribosomal protein, laminin-binding protein, and human lymphocyte antigen class I messenger RNAs associated with colon carcinoma progression and differentiation."
06/01/2005 - "The 20 genes with at least a 3-fold change, annotated with known phenotypic associations in the current gene databank (phenotype association, fold change) were aspartoacylase (Canavan disease, 9.96), growth hormone receptor (Laron dwarfism, idiopathic short stature, 8.25), lipoprotein lipase (familial chylomicronemia syndrome, lipoprotein lipase deficiency, 8.00), vitamin D (1,25- dihydroxyvitamin D3) receptor (involutional osteoporosis, vitamin D resistant rickets, 7.94), intercellular adhesion molecule 1 human rhinovirus receptor (cerebral malaria susceptibility, 7.16), peroxisomal membrane protein 3 35-kDa (Refsum disease, infantile form, Zellweger syndrome-3, 6.00), Bardet-Biedl syndrome 2 (Bardet-Biedl syndrome, 5.87), ribosomal protein S19 (Diamond Blackfan anemia, 5.85), apolipoprotein C-III (hypertriglyceridemia, 5.44), argininosuccinate lyase (argininosuccinicaciduria, 5.22), myosin VA (Griscelli syndrome-type pigmentary dilution with mental retardation, 4.92), lysozyme (renal amyloidosis, 4.17), SAM domain, SH3 domain and nuclear localisation signals 1 (Cherubism, 4.12 ), von Hippel-Lindau syndrome (hemangioblastoma, cerebellar, somatic, von Hippel-Lindau syndrome, 3.94), early-onset breast cancer 1 (BRCA1, papillary serous carcinoma of the peritoneum, 3.73), UDP-N-acetylglucosamine-2-epimerase/N-acetylmannosamine kinase (inclusion body myopathy, autosomal recessive, sialuria, 3.53), apolipoprotein A-I (amyloidosis, 3 or more types, hypoalphalipoproteinemia, 3.29), midline 1 Opitz/BBB syndrome (Opitz G syndrome, type I, 3.28), ATPase, Na+/K+ transporting, alpha 2 (+) polypeptide (familial hemiplegic migraine, 3.05). "
|1.||Proteasome Endopeptidase Complex (Proteasome)
|2.||Small Interfering RNA (siRNA)
|3.||Messenger RNA (mRNA)
|5.||Janus Kinase 2
|7.||TNF-Related Apoptosis-Inducing Ligand Receptors
|8.||5' Untranslated Regions (5' UTR)
|10.||Intercellular Adhesion Molecule-1 (Intercellular Adhesion Molecule 1)
|1.||Transplantation (Transplant Recipients)