|1.||Nishie, Wataru: 17 articles (05/2015 - 03/2007)|
|2.||Bruckner-Tuderman, Leena: 17 articles (05/2015 - 01/2002)|
|3.||Shimizu, Hiroshi: 14 articles (05/2015 - 03/2007)|
|4.||Sitaru, Cassian: 14 articles (04/2014 - 01/2002)|
|5.||Tasanen, Kaisa: 13 articles (05/2015 - 03/2003)|
|6.||Zillikens, Detlef: 12 articles (08/2014 - 01/2002)|
|7.||Ujiie, Hideyuki: 9 articles (05/2015 - 09/2009)|
|8.||Natsuga, Ken: 7 articles (05/2015 - 01/2009)|
|9.||Franzke, Claus-Werner: 6 articles (05/2015 - 04/2008)|
|10.||Sawamura, Daisuke: 6 articles (04/2015 - 03/2007)|
|1.||Bullous Pemphigoid (Pemphigoid)
02/01/2011 - "Type XVII collagen ELISA indices significantly decreased after bullous pemphigoid remission."
05/01/2012 - "In vitro studies on ectodomain shedding of type XVII collagen have also provided basic knowledge on the development of bullous pemphigoid. "
07/01/2011 - "In vitro studies on ectodomain shedding of type XVII collagen have also provided basic knowledge on the development of bullous pemphigoid. "
03/01/2014 - "Update on the pathogenesis of bullous pemphigoid: an autoantibody-mediated blistering disease targeting collagen XVII."
01/01/2013 - "In addition, collagen XVII has been found to be present in the central nervous system, thus offering an explanation for the statistical association between bullous pemphigoid, in which autoimmunity is directed against dermal collagen XVII, and neurological diseases. "
03/01/2015 - "Increased collagen XVII expression was associated with decreased disease-free and cancer-specific survival. "
05/01/2006 - "The expression of collagen XVII (BP180), a transmembrane hemidesmosomal component, is upregulated in invasive areas of epithelial tumors. "
07/01/2003 - "These results indicate altered expression of collagen XVII at different stages of carcinogenesis and suggest a correlation between overexpression of collagen XVII and tumor progression. "
07/01/2003 - "Overexpression of collagen XVII was found at the invasive front of the tumors. "
11/01/2001 - "Diffuse intracellular distribution of collagen XVII and a consequent loss of critical cellular attachments may contribute to the infiltrative and progressive growing potential of tumors."
02/01/2000 - "Despite a number of studies on autoantibody targets in blistering skin diseases, it has remained unclear whether the physiologically shed ectodomain of collagen XVII plays a role as an autoantigen. "
02/01/2000 - "Sera of 81 patients with a clinically active blistering skin disease were reacted with full-length collagen XVII, the authentic soluble ectodomain, and recombinant fragments. "
04/01/2015 - "Bullous pemphigoid (BP) is a common autoimmune blistering skin disease in which two hemidesmosomal components--the transmembrane collagen XVII (BP180 or BPAG2) and the plakin family protein BP230 (BPAG1)--are targeted by autoimmunity. "
03/01/2007 - "Bullous pemphigoid (BP) is the most common serious autoimmune blistering skin disease, with a considerable body of indirect evidence indicating that the underlying autoantigen is collagen XVII (COL17). "
10/01/2012 - "Inherited mutations or auto-antibodies targeting collagen XVII cause blistering skin disease. "
|4.||Junctional Epidermolysis Bullosa (Epidermolysis Bullosa Letalis)
01/01/2001 - "In this study we describe six Italian patients presenting an unusually mild variant of non-Herlitz junctional epidermolysis bullosa associated with a reduced expression of type XVII collagen. "
05/01/2012 - "Mutations in the type XVII collagen gene (COL17A1) result in the blistering disorder non-Herlitz junctional epidermolysis bullosa (JEB-nH). "
01/01/2010 - "Collagen XVII has been identified as having a role in inherited junctional epidermolysis bullosa non-Herlitz (JEB-other, MIM #226650). "
09/01/2009 - "Collagen XVII (COL17) participates in keratinocyte adhesion and possibly migration, as COL17 defects disrupt keratinocyte-basal lamina adhesion and underlie the disease non-Herlitz junctional epidermolysis bullosa. "
05/01/2007 - "Mutations in the gene COL17A1 coding for type XVII collagen cause non-Herlitz junctional epidermolysis bullosa (nH-JEB). "
02/01/2002 - "Generalized atrophic benign epidermolysis bullosa [GABEB (OMIM no. 226650)] is an inherited subepidermal blistering disease typically caused by null mutations in COL17A1, the gene encoding type XVII collagen. "
06/01/2001 - "However, in our patient with the generalized atrophic benign epidermolysis bullosa subtype, a small amount of type XVII collagen was detectable in the skin, and immunoblotting of cultured keratinocytes revealed that the 180-kDa protein was 10 kDa shorter. "
02/01/1998 - "Patients with generalized atrophic benign epidermolysis bullosa often show decreased expression of type XVII collagen, a transmembrane hemidesmosomal protein encoded by COL17A1. "
08/01/1997 - "Implications of intragenic marker homozygosity and haplotype sharing in a rare autosomal recessive disorder: the example of the collagen type XVII (COL17A1) locus in generalised atrophic benign epidermolysis bullosa."
02/01/1997 - "Cloning of the human type XVII collagen gene (COL17A1), and detection of novel mutations in generalized atrophic benign epidermolysis bullosa."
|3.||Immunoglobulin A (IgA)
|4.||Messenger RNA (mRNA)
|5.||Immunoglobulin G (IgG)
|6.||Proteins (Proteins, Gene)
|10.||Collagen Type VII
|1.||Heterologous Transplantation (Xenotransplantation)