|1.||Rastogi, Nalin: 3 articles (01/2012 - 08/2005)|
|2.||Lu, Chuan-Jian: 1 article (05/2012)|
|3.||Li, Sha-Sha: 1 article (05/2012)|
|4.||Jiang, Zhi-Ting: 1 article (05/2012)|
|5.||Lahlou, Ouafae: 1 article (01/2012)|
|6.||Chaoui, Imane: 1 article (01/2012)|
|7.||Millet, Julie: 1 article (01/2012)|
|8.||El Aouad, Rajae: 1 article (01/2012)|
|9.||Akrim, Mohammed: 1 article (01/2012)|
|10.||Filali-Maltouf, Abdelkarim: 1 article (01/2012)|
01/01/2012 - "tuberculosis population structure in Morocco is highly homogeneous, and is characterized by the predominance of the Euro-American lineages, namely LAM, Haarlem, and T, which belong to the "evolutionary recent" TbD1-/PGG2/3 phylogenetic group. "
12/01/2007 - "tuberculosis populations; however, the majority of the ongoing transmission is due to "evolutionary recent" tuberculosis lineages belonging to principal genetic group 2 (PGG2; Haarlem and LAM) and PGG3 (ill-defined T clade), and most of it is attributable to the LAM7-TUR sublineage with an enhanced phylogeographical specificity for Turkey. "
08/01/2005 - "tuberculosis isolates of the PGG2 and PGG3 groups, such as Haarlem, Latin-American and Mediterranean (LAM), and X families. "
01/01/1981 - "In this respect Kuehl et al. proposed the most interesting idea that a true mediator of inflammation is PGG2 owing to the presence of 15-hydroperoxy group in its molecule, and its capability to generate either TXA2 or free hydroxy radical. "
12/01/1977 - "However, direct evidence is presented to show that an oxygen equivalent released in the enzymatic conversion of PGG2 to PGH2 is a prime factor in inflammation."
05/01/2012 - "Results There was no statistical difference in the potential biomarkers contents correlated to symptoms of fever, inflammation and cough, such as PGG2, 20-COOH-LTB4, homocystein, and so on in the serum of patients of various syndrome types before treatment (P > 0.05). "
12/01/1977 - "That both MK-447 and prostaglandin synthetase inhibitors depress the enzymatic formation of PGG2 from arachidonic acid suggests that this endoperoxide plays a pivotal role in acute inflammation. "
11/01/2010 - "TxA2, F2-isoprostanes, prostaglandins endoperoxides PGG2/PGH2) are known to promote the initiation and progression of atherosclerosis, not only via platelet activation, but through leukocyte-endothelial interactions and vasoconstriction. "
01/01/2001 - "Endothelial dysfunction in patients suffering from atherosclerosis or diabetes type 2 is associated not only with suppression in release of the above mediators but also with deleterious discharge of prostaglandin endoperoxides (PGH2, PGG2), superoxide anion (O2-, peroxynitrite (ONOO-), and plasminogen activator inhibitor (PAI-1). "
|5.||Platelet Storage Pool Deficiency
|1.||Prostaglandin H2 (PGH(2))
|2.||Arachidonic Acid (Vitamin F)
|4.||Biological Markers (Surrogate Marker)
|6.||Cyclooxygenase 2 (Cyclooxygenase-2)
|7.||Peroxynitrous Acid (Peroxynitrite)
|8.||F2-Isoprostanes (F2 Isoprostanes)