|1.||Calixto, João B: 4 articles (07/2013 - 04/2005)|
|2.||Chaves, Mariana H: 3 articles (07/2011 - 10/2005)|
|3.||Santos, F A: 3 articles (08/2008 - 12/2007)|
|4.||Chaves, M H: 3 articles (08/2008 - 12/2007)|
|5.||Bento, Allisson Freire: 2 articles (07/2013 - 08/2011)|
|6.||Marcon, Rodrigo: 2 articles (07/2013 - 10/2009)|
|7.||Das, Pijush K: 2 articles (05/2012 - 07/2005)|
|8.||Santos, Flávia A: 2 articles (07/2011 - 01/2006)|
|9.||Rao, V S: 2 articles (08/2008 - 02/2008)|
|10.||Cunha, G M A: 2 articles (08/2008 - 02/2008)|
09/14/2010 - "To study the beneficial effects of triterpene alpha,beta-amyrin and the underlying mechanisms in an experimental pancreatitis model. "
09/14/2010 - "alpha,beta-amyrin has the potential to combat acute pancreatitis by acting as an anti-inflammatory and antioxidant agent."
07/01/2011 - "α,β-Amyrin ameliorates cerulein-induced acute pancreatitis by acting as an anti-inflammatory and antioxidant agent."
09/14/2010 - "alpha,beta-amyrin, a natural triterpenoid ameliorates L-arginine-induced acute pancreatitis in rats."
07/01/2011 - "To evaluate the anti-inflammatory effect of α,β-amyrin, a pentacyclic triterpenoid from Protium heptaphyllum, on cerulein-induced acute pancreatitis in mice. "
|2.||Body Weight (Weight, Body)
02/23/2015 - "Experimental rats, hepato-oxidatively stressed by CCl4 (0.2 ml/kg b wt/twice a week, intra-peritoneally), were concurrently received α-amyrin (20mg/kg body weight/day, orally) for 30 consecutive days. "
02/23/2015 - "The assessment of all biochemical parameters registered a significant (P<0.001) hepatic oxidative stress in CCl4 treated rats, which was considerably recovered near to almost normal level in rats co-administered with α-amyrin at the dose level of 20mg/kg body weight/day for 30 consecutive days. "
07/01/2013 - "Our results showed that the oral preventive or therapeutic treatment with α,β-amyrin significantly reduced disease activity, body weight loss, colonic damage, as well as colonic myeloperoxidase and N-acetylglucosaminidase activities. "
09/14/2010 - "alpha,beta-amyrin and methylprednisolone treatments significantly (P < 0.05) attenuated the L-arginine-induced increases in pancreatic wet weight/body weight ratio, and decreased the serum levels of amylase and lipase, and TNF-alpha and IL-6, as compared to the vehicle control. "
|3.||Vascular Diseases (Vascular Disease)
07/01/2009 - "Here, we evaluated the effects of a 1:1 mixture of alpha- and beta-amyrin (alpha,beta-amyrin) on an experimental model of colitis in mice. "
07/01/2013 - "Interestingly, our results also showed that the cannabinoid receptor 1 (CB(1)), but not CB(2), pharmacological blockade significantly reversed the beneficial effects of α,β-amyrin in DSS-induced colitis. "
07/01/2013 - "We assessed the anti-inflammatory effects of the α,β-amyrin in the dextran sulfate sodium (DSS)-induced colitis in mice and investigated whether its effects were associated with the interaction with the cannabinoid system. "
07/01/2009 - "Colitis was induced in Swiss male mice by trinitrobenzene sulphonic acid (TNBS) and followed up to 72 h; animals were treated systemically with alpha,beta-amyrin, dexamethasone or vehicle. "
07/01/2009 - "Therapeutic action and underlying mechanisms of a combination of two pentacyclic triterpenes, alpha- and beta-amyrin, in a mouse model of colitis."
|7.||Cannabinoid Receptors (Cannabinoid Receptor)
|9.||Tumor Necrosis Factor-alpha (Tumor Necrosis Factor)