|1.||Domino, Karen B: 2 articles (05/2010 - 08/2007)|
|2.||Haberkern, Charles M: 2 articles (05/2010 - 08/2007)|
|3.||Campos, John S: 2 articles (05/2010 - 08/2007)|
|4.||Bhananker, Sanjay M: 2 articles (05/2010 - 08/2007)|
|5.||Posner, Karen L: 2 articles (05/2010 - 08/2007)|
|6.||Ramamoorthy, Chandra: 2 articles (05/2010 - 08/2007)|
|7.||Morray, Jeffrey P: 2 articles (05/2010 - 08/2007)|
|8.||Ramamoorthy, C: 2 articles (09/2002 - 07/2000)|
|9.||Haberkern, C M: 2 articles (09/2002 - 07/2000)|
|10.||Morray, J P: 2 articles (09/2002 - 07/2000)|
01/01/1988 - "In hearts of CD rats insulin resistance is not dependent on disturbances in lipid metabolism and is practically not influenced by POCA. "
01/01/1988 - "In hearts of AD rats an absolute insulin resistance was observed which could be attenuated by perfusion of the hearts with POCA (10 microM). "
01/01/1988 - "The insulin resistance can be eliminated and has to be distinguished from a defect in the glucose uptake system not affected by POCA. "
01/01/1988 - "In hearts of CD rats, which show a relative insulin resistance, POCA only slightly stimulated glucose oxidation and uptake, but the total rate of uptake and conversion of glucose as well as the responsiveness of these hearts to insulin remained low. "
02/01/1984 - "(2) Since in vitro perfusion with POCA partially restored the insulin sensitivity of the diabetic hearts, insulin-receptor defects should be of minor importance for the insulin resistance of diabetic hearts. "
03/01/1989 - "The present study was performed to determine whether POCA could prevent accumulation of both LCA and LPC induced by ischemia in vivo and if so, whether attenuation of early arrhythmogenesis would result. "
12/01/1991 - "In conclusion, during transient ischemia POCA and pyruvate markedly increased cardiac FA accumulation through inhibition of the oxidation of FAs released from endogenous lipid pools. "
09/01/1988 - "However, during ischemia, it is likely that the depletion of ATP concentration induced by POCA resulted in delayed recovery of mechanical function on reperfusion."
09/01/1988 - "During the ischemic period, though, POCA decreased the ATP concentration at a rate three times that of controls during the first 10 min. No further reductions were observed for up to 30 min of ischemia. "
03/01/1986 - "POCA may protect the ischemic heart by preventing accumulation of these toxic metabolites and by stimulating glucose utilization during ischemia."
|4.||Body Weight (Weight, Body)
01/01/1988 - "The specific carnitine palmitoyltransferase I (CPT I)-inhibitor POCA - sodium-2(5-(4-chlorphenyl)pentyl-oxirane carboxylate - was used in isolated perfused hearts of acutely diabetic, ketotic (AD, 100 mg streptozotocin/kg body weight), chronically diabetic (CD, 60 mg streptozotocin/kg body weight), and obese ZUCKER rats (fa/fa) to study different forms of insulin resistance. "
|5.||Heart Arrest (Cardiac Arrest)
09/01/2002 - "This report provides a brief historical account of the development of the Pediatric Peri-Operative Cardiac Arrest (POCA) Registry and elaborates on the methodology including strengths, weaknesses, and practical implementation issues."
09/01/2002 - "Pediatric Peri-Operative Cardiac Arrest (POCA) Registry."
07/01/2000 - "In the first 4 yr of the POCA Registry, 63 institutions enrolled and submitted 289 cases of cardiac arrest. "
07/01/2000 - "The Pediatric Perioperative Cardiac Arrest (POCA) Registry was formed in 1994 in an attempt to determine the clinical factors and outcomes associated with cardiac arrest in anesthetized children. "
07/01/2000 - "Anesthesia-related cardiac arrest in children: initial findings of the Pediatric Perioperative Cardiac Arrest (POCA) Registry."
|2.||Carnitine O-Palmitoyltransferase (Carnitine Palmitoyltransferase II)
|4.||Ethylene Oxide (Oxirane)
|6.||7- beta- galactopyranosyloxycoumarin- 4- acetic acid
|10.||Pyruvic Acid (Pyruvate)