|1.||Gao, Ling-Juan: 4 articles (02/2012 - 11/2011)|
|2.||Gu, Ping-Qing: 4 articles (02/2012 - 11/2011)|
|3.||Liu, Zhu: 3 articles (02/2012 - 11/2011)|
|4.||Gu, Ping-qing: 2 articles (01/2014 - 01/2013)|
|5.||Gao, Ling-juan: 2 articles (01/2014 - 01/2013)|
|6.||Guo, Xi-Rong: 2 articles (02/2012 - 11/2011)|
|7.||Peng, Yu-Zhu: 2 articles (02/2012 - 11/2011)|
|8.||Su, Ya-Juan: 2 articles (01/2012 - 01/2012)|
|9.||Su, Ya-juan: 1 article (01/2014)|
|10.||Chen, Zheng-lin: 1 article (01/2014)|
|1.||Systemic Lupus Erythematosus (Libman-Sacks Disease)
10/01/2006 - "C1qRP (CD93) expression on peripheral blood monocytes in patients with systemic lupus erythematosus."
01/01/2004 - "Role of C1q and C1q receptors in the pathogenesis of systemic lupus erythematosus."
10/01/2006 - "Aim of the study was to compare the expression of monocytes C1qRp (CD93) in patients with systemic lupus erythematosus (SLE) with that of healthy controls and to determine the influence of glucocorticoids and LPS on C1qRp expression. "
|3.||Spontaneous Abortion (Miscarriage)
|4.||Squamous Cell Carcinoma (Epidermoid Carcinoma)
01/01/2014 - "The role of the globular heads of the C1q receptor in HPV-16 E2-induced human cervical squamous carcinoma cell apoptosis via a mitochondria-dependent pathway."
01/01/2012 - "The globular heads of the C1q receptor regulate apoptosis in human cervical squamous carcinoma cells via a p53-dependent pathway."
01/01/2012 - "The role of the globular heads of C1q receptor (gC1qR) gene in regulating apoptosis of human cervical squamous cell carcinoma."
01/01/2013 - "The role of globular heads of the C1q receptor in HPV 16 E2-induced human cervical squamous carcinoma cell apoptosis is associated with p38 MAPK/JNK activation."
04/01/1992 - "In injury and inflammation, interactions of complement C1q with C1q receptors may provide attachment sites for cell localization and tissue regeneration. "
04/01/1992 - "Our results indicate that C1q binding and the type of C1q receptors can serve as markers for fibroblast subpopulations differing in collagen synthesis, and that selection of subpopulations and their differential sensitivity to regulatory molecules can contribute to collagen alterations associated with inflammation, fibrosis, and other acquired diseases."
|1.||Polychlorinated Biphenyls (PCBs)
|3.||Complement C1q (C1q Complement)
|4.||p38 Mitogen-Activated Protein Kinases
|5.||Interleukin-8 (Interleukin 8)
|8.||Antigen-Antibody Complex (Immune Complex)
|1.||Induced Abortion (Induced Abortions)