|1.||Jiann, Bang-Ping: 2 articles (06/2003 - 02/2002)|
|2.||Chen, Wei-Chung: 2 articles (06/2003 - 02/2002)|
|3.||Jan, Chung-Ren: 2 articles (06/2003 - 02/2002)|
|4.||Huang, Jong-Khing: 2 articles (06/2003 - 02/2002)|
|5.||Lu, Yih-Chau: 2 articles (06/2003 - 02/2002)|
|6.||Chang, Hong-Tai: 2 articles (06/2003 - 02/2002)|
|7.||Sparatore, Fabio: 2 articles (11/2002 - 07/2002)|
|8.||Bruzzo, Cristina: 2 articles (11/2002 - 07/2002)|
|9.||Schettini, Gennaro: 2 articles (11/2002 - 07/2002)|
|10.||Alama, Angela: 2 articles (11/2002 - 07/2002)|
|1.||Brain Edema (Cerebral Edema)
07/01/1986 - "[Cerebral edema induced by triethyltin chloride in the rat: applications to the study of anti-cerebral edema substances]."
05/01/1984 - "In golden hamsters, a study was made on the vascular permeability changes which might take place during the formation of triethyltin (TET)-induced brain edema. "
05/01/2007 - "In this model, daily oral triethyltin (TET) administration for 5 consecutive days strongly perturbed the rat behaviour and induced a cerebral edema at the 5th day. "
06/09/1995 - "Brain edema was produced by injecting triethyltin (TET) into the right cerebral hemisphere via the internal carotid artery in rats. "
01/01/1991 - "Triethyltin (TET) is a neurotoxicant that produces severe but transient cerebral edema, characterized ultrastructurally by vacuolation of the intraperiod line of central nervous system (CNS) myelin. "
07/13/1979 - "A sequential study of changes in the brain and cerebrospinal fluid of the rat following triethyltin poisoning."
01/01/1988 - "[Implication of lipid peroxidation in triethyltin poisoning in the rat]."
10/01/1987 - "Acute poisoning with triethyltin in the rat. "
01/01/1987 - "[Changes in ECoG and ECG in acute triethyltin poisoning in rats]."
01/01/1969 - "Collapse of the blood-brain barrier in lymphostatic cerebral hemangiopathy and in triethyltin poisoning."
12/01/1996 - "Compound 22 shows protective effects against hypobaric hypoxia and triethyltin induced brain edema."
03/01/1982 - "Acute administration of triethyltin (TET) produces a well-described sequence of pathological events characterized by intramyelinic vacuolation, edema, and histotoxic hypoxia. "
12/01/1981 - "In adults, triethyltin (TET) produces degeneration of white matter, edema, vacuolization on myelin and histotoxic hypoxia. "
01/01/2011 - "One such "marker" is cell swelling; it occurs in response to a diversity of insults, such as physical damage, disease (ischemia, trauma, and hypoxia), and chemicals (methylmercury, lead, 1,3-dinitrobenzene, and triethyltin). "
|4.||Demyelinating Diseases (Demyelinating Disease)
10/01/1978 - "For initial study, triethyltin was chosen as an agent producing demyelination of nerves, acrylamide as an agent producing "dying-back" neuropathy, and methylmercury as an agent producing mixed central and peripheral neuropathies. "
08/01/1999 - "Whereas trimethyltin (TMT) is known to induce gliosis and triethyltin (TET) to cause demyelination and affect GABAergic neurons, DBT appeared to be more toxic than TMT, and to present a distinct toxicological pattern."
01/01/1989 - "There was no evidence of demyelination, axonal degeneration, or damage uolation was histologically similar to that observed in association with other drugs such as triethyltin, isoniazid, or hexachlorophene. "
12/01/2002 - "In vivo studies have indicated that systemically administered bilobalide, a sesquiterpene trilactone constituent of Ginkgo biloba leaf extracts, can reduce cerebral edema produced by triethyltin, decrease cortical infarct volume in certain stroke models, and reduce cerebral ischemia. "
|3.||barbituric acid (barbiturate)