triethyltin

05/01/1984 - "In golden hamsters, a study was made on the vascular permeability changes which might take place during the formation of triethyltin (TET)-induced brain edema. "
05/01/2007 - "In this model, daily oral triethyltin (TET) administration for 5 consecutive days strongly perturbed the rat behaviour and induced a cerebral edema at the 5th day. "
06/09/1995 - "Brain edema was produced by injecting triethyltin (TET) into the right cerebral hemisphere via the internal carotid artery in rats. "
01/01/1991 - "Triethyltin (TET) is a neurotoxicant that produces severe but transient cerebral edema, characterized ultrastructurally by vacuolation of the intraperiod line of central nervous system (CNS) myelin. "
01/01/1986 - "Effect of an extract of Ginkgo biloba on triethyltin-induced cerebral edema."

07/13/1979 - "A sequential study of changes in the brain and cerebrospinal fluid of the rat following triethyltin poisoning."
01/01/1988 - "[Implication of lipid peroxidation in triethyltin poisoning in the rat]."
10/01/1987 - "Acute poisoning with triethyltin in the rat. "
01/01/1987 - "[Changes in ECoG and ECG in acute triethyltin poisoning in rats]."
01/01/1969 - "Collapse of the blood-brain barrier in lymphostatic cerebral hemangiopathy and in triethyltin poisoning."

12/01/1996 - "Compound 22 shows protective effects against hypobaric hypoxia and triethyltin induced brain edema."
03/01/1982 - "Acute administration of triethyltin (TET) produces a well-described sequence of pathological events characterized by intramyelinic vacuolation, edema, and histotoxic hypoxia. "
12/01/1981 - "In adults, triethyltin (TET) produces degeneration of white matter, edema, vacuolization on myelin and histotoxic hypoxia. "
01/01/2011 - "One such "marker" is cell swelling; it occurs in response to a diversity of insults, such as physical damage, disease (ischemia, trauma, and hypoxia), and chemicals (methylmercury, lead, 1,3-dinitrobenzene, and triethyltin). "

05/01/2004 - "Triethyltin (TET)-induced neurotoxicity in the brain causes the formation of myelin edema and loss. "
01/01/2000 - "Lessons learned in the mechanistic investigation of neurotoxicants suggest caution in the evaluation and interpretation of structure-activity relationships, eg, TMT, MeHg, and DNB all induce oxidative stress, whereas TMT and triethyltin produce neuronal damage and myelin edema, respectively. "
03/01/1989 - "To better understand the role of myelin-associated water in the differentiation of white and gray matter in magnetic resonance (MR) imaging, changes in MR relaxation processes were studied in rabbits during myelination and after induction of cytotoxic edema with triethyltin (TET). "
01/01/1983 - "For this purpose, we produced in the mouse an acute cytotoxic edema by triethyltin intoxication and a vasogenic edema by a cortical cryogenic injury. "
12/01/1979 - "Brain stem auditory evoked potentials and myelin changes in triethyltin-induced edema in young adult rats."

10/01/1978 - "For initial study, triethyltin was chosen as an agent producing demyelination of nerves, acrylamide as an agent producing "dying-back" neuropathy, and methylmercury as an agent producing mixed central and peripheral neuropathies. "
08/01/1999 - "Whereas trimethyltin (TMT) is known to induce gliosis and triethyltin (TET) to cause demyelination and affect GABAergic neurons, DBT appeared to be more toxic than TMT, and to present a distinct toxicological pattern."
01/01/1989 - "There was no evidence of demyelination, axonal degeneration, or damage uolation was histologically similar to that observed in association with other drugs such as triethyltin, isoniazid, or hexachlorophene. "