|1.||Banik, Bimal K: 2 articles (03/2010 - 05/2006)|
|2.||Becker, Frederick F: 2 articles (03/2010 - 05/2006)|
|3.||Pogrmic-Majkic, Kristina: 1 article (01/2015)|
|4.||Navarro, Patricio: 1 article (01/2015)|
|5.||Álvarez-León, Eva E: 1 article (01/2015)|
|6.||Lübcke-von Varel, Urte: 1 article (01/2015)|
|7.||Fa, Svetlana: 1 article (01/2015)|
|8.||Almeida-González, Maira: 1 article (01/2015)|
|9.||Boada, Luis D: 1 article (01/2015)|
|10.||Kaisarevic, Sonja: 1 article (01/2015)|
01/01/2012 - "Two "ED(001)" cancer studies have been conducted, utilizing approximately 40,000 trout, by dietary exposure to AFB(1) and dibenzo[d,e,f,p]chrysene (DBC). "
11/01/1994 - "5-MeC was found to be a potent lung carcinogen in strain A/J mice, inducing more than 100 tumors/mouse at a concentration of 200 mg/kg. Six 5-MeC-DNA adducts were observed; one adduct comigrated with the standard N2-deoxyguanosine adduct of 5-MeC-diol-epoxide I [1R,2S,3S-trihydroxy-4R-(N2-deoxy-guanosyl-3'-phosphate)- 1,2,3,4-tetrahydro-5-methyl-chrysene], derived from the bay-region diol-epoxide of 5-MeC. "
12/01/1988 - "4,5-Methylenechrysene elicited twice as many tumors as chrysene at each of the doses tested. "
08/01/1986 - "Chrysene administered at 2800 nmol per mouse induced hepatic and lung tumors in 41% and 21% of the males, respectively; at the 700-nmol dose, it induced only liver tumors in 29% of the males and in none of the females. "
06/01/1978 - "The most potent tumor initiator was chrysene 1,2-dihydrodiol, which had approximately twice the tumorigenic activity of the parent hydrocarbon chrysene at all doses tested. "
|3.||Hepatocellular Carcinoma (Hepatoma)
01/01/2015 - "Rat hepatoma cells H4IIE were treated by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and polycyclic aromatic hydrocarbons (PAHs) (dibenz(a,h)anthracene, benzo(a)pyrene, benz(a)anthracene, chrysene), low-concentration mixtures of PAHs and TCDD, and environmental mixtures contaminated by PAHs and their derivatives. "
07/07/2012 - "To evaluate the change in gene expression levels, human hepatocellular carcinoma (HepG2) cells were exposed to nine different PAHs (benzo[a]pyrene, dibenzo[a,h]anthracene, 3-methylcholanthrene, naphthalene, chrysene, phenanthrene, benzo[a]anthracene, benzo[k]fluoranthene, and indeno[1,2,3-c,d]pyrene) for 48 h. "
06/01/2002 - "Using this quantitative structure-activity relationship (QSAR) to calculate threshold values for the toxicity of the remaining nontoxic substances (benz[a]anthracene, chrysene, benzo[b]fluoranthene, benzo[k]fluoranthene, dibenz[a,h]anthracene, benzo[a]pyrene, perylene, and indeno[1,2,3-cd]pyrene), the absence of toxicity could, in most cases, be explained by a limited water solubility, indicating that these substances do act by narcosis as the mode of toxic action and that their toxicity is governed by concentrations in the pore-water. "
|5.||Photosensitivity Disorders (Photodermatitis)