|1.||Harrington, Chris F: 1 article (03/2015)|
|2.||Taylor, Andrew: 1 article (03/2015)|
|3.||Hong, Estrella: 1 article (09/2013)|
|4.||Whitesides, George M: 1 article (09/2013)|
|5.||Derda, Ratmir: 1 article (09/2013)|
|6.||Mazzeo, Aaron: 1 article (09/2013)|
|7.||Ingber, Donald E: 1 article (09/2013)|
|8.||Deiss, Frédérique: 1 article (09/2013)|
|9.||Sela, Shifra: 1 article (11/2007)|
|10.||Mazor, Rafi: 1 article (11/2007)|
|1.||Acute Myeloid Leukemia (Acute Myelogenous Leukemia)
10/15/1999 - "To test the hypothesis that arsenic compounds may be effective against other forms of acute myelogenous leukemia (AML), we studied the membrane-permeable arsenic compound phenylarsine oxide (PAO). "
10/15/1999 - "Phenylarsine oxide blocks interleukin-1beta-induced activation of the nuclear transcription factor NF-kappaB, inhibits proliferation, and induces apoptosis of acute myelogenous leukemia cells."
02/01/2004 - "Unlike previous analogous studies with 4-N-(S-glutathionylacetyl)amino) phenylarsenoxide (GSAO)-Cy5.5 in which the tumor image faded with time, the endostatin-Cy5.5 NIR signal was emitted from the tumor up to 7 days after injection, the last time point examined. "
03/15/2015 - "One 4-(N-(S-glutathionylacetyl)amino) phenylarsenoxide (GSAO) containing arsenic and under investigation for the treatment of solid tumours, and the second cis-diamminedichloroplatinum (II) (cisplatin) containing platinum and widely used in the clinical setting as a front line treatment against various neplasias in particular testicular, ovarian, bladder and head and neck cancers. "
|3.||Glioblastoma (Glioblastoma Multiforme)
06/01/2001 - "With respect to phosphatases, phenylarsine oxide significantly increased perfusion pressure in normoxia as well as in hypoxia. "
11/01/1991 - "I: Effects of anoxia, phenylarsine oxide and monensin."
05/21/2004 - "The NADPH oxidase inhibitors diphenylene iodonium (10microM) and phenylarsine oxide (5microM) were without effect on either basal Ca(2+) currents or responses to hypoxia. "
10/26/2001 - "For comparison, LLC-PK(1) cells were exposed to hypoxia, stimulated with desferroxamine (DFX, known to mimic hypoxia), and the thiol-cross-linking agent phenylarsine oxide (PAO). "
03/01/1999 - "Neopterin (3-300 microM) and phenylarsine oxide (0.5-2 microM), other inhibitors of NADPH oxidase, did not promote release of [3H]CA in normoxic conditions or affect the response elicited by hypoxia. "
11/01/1992 - "The heavy metal Hg2+ and heat shock stimulated PTPase activity at an optimal dose, whereas the oxidant phenylarsine oxide (PAO) was only marginally stimulatory. "
02/15/1991 - "AII-induced internalization was not influenced by the presence or absence of extracellular Ca2+ but was inhibited by treatment with phenylarsine oxide (PAO) or by arresting coated pit formation with hypotonic shock and potassium depletion. "
10/01/1992 - "AG126 enhanced IFN-gamma, IL-1, and IL-6 production in PBM that were costimulated with the stress stimuli heat shock and phenylarsine oxide. "
12/15/1999 - "Induction of heme oxygenase-1 can be caused by numerous factors, including heme, other metalloporphyrins, transition metal ions, heat shock, ultraviolet light, phorbol esters, sodium arsenite, and phenylarsine oxide (PAO). "
07/01/1997 - "Modulation of signal components of this apoptotic pathway resulted the following a) phorbol ester (PMA) or heat shock inhibited apoptosis, which was prevented by staurosporine b) 3-amino-benzamide, a potent poly(ADP-ribose)polymerase inhibitor, had no significant effect; c) cysteine reactive compounds, such as iodoacetamide and phenylarsine oxide, as well as protease inhibitor TPCK were very active inhibitors of apoptosis; d) protein synthesis inhibitor, cycloheximide, potentiated cell death; e) the ETO-induced p53 protein overexpression had neither enhancing nor protecting effect on the apoptotic process. "
|2.||Arsenicals (Arsenic Compounds)
|3.||Protein Tyrosine Phosphatases
|5.||Matrix Metalloproteinase 9 (Gelatinase B)
|7.||CY5.5 cyanine dye
|8.||NADPH Oxidase (NAD(P)H oxidase)
|9.||Phosphoric Monoester Hydrolases (Phosphatases)