|1.||Volpi, Riccardo: 2 articles (12/2011 - 01/2004)|
|2.||Coiro, Vittorio: 2 articles (12/2011 - 01/2004)|
|3.||Chiodera, Paolo: 2 articles (12/2011 - 01/2004)|
|4.||Obiero, Josiah M: 1 article (12/2015)|
|5.||Nickell, Justin R: 1 article (12/2015)|
|6.||Sviripa, Vitaliy M: 1 article (12/2015)|
|7.||Burikhanov, Ravshan: 1 article (12/2015)|
|8.||Zhan, Chang-Guo: 1 article (12/2015)|
|9.||Liu, Chunming: 1 article (12/2015)|
|10.||Rangnekar, Vivek M: 1 article (12/2015)|
|1.||Hypoglycemia (Reactive Hypoglycemia)
03/01/1986 - "In this study, we investigated the importance of putative forebrain glucoreceptors to the systemic response to hypoglycemia. "
12/10/2011 - "Glucoreceptors located in the brain mediate NPY release induced by hypoglycemia in normal men."
01/01/2004 - "Glucoreceptors located inside the blood-brain barrier mediate hypoglycemia-induced LH inhibition in man."
01/01/1991 - "Utilizing this approach, there appear to be no glucoreceptors residing exclusively in either the forebrain or hindbrain which are essential for the sympathoadrenal response to hypoglycemia. "
01/01/1991 - "To elucidate the loci for the putative glucoreceptors responding to hypoglycemia we introduced 'brain' and 'liver' clamps. "
04/01/2004 - "Glucose-receptor MR imaging of tumors: study in mice with PEGylated paramagnetic niosomes."
02/01/2014 - "γ-Fe2O3@DMSA-DG NPs has an ability to target glucose receptors overexpressed in tumors, and may serve as a MRI contrast agent for tumor detection."
12/15/2015 - "Prostate apoptosis response-4 protein (Par-4) is a tumor suppressor that causes apoptosis in therapy-resistant prostate cancer cells by binding specifically to a receptor, Glucose-regulated protein-78 (GRP78), found only on the surface of cancer cells. "
01/01/1986 - "We have investigated the binding efficiency and shown that: 3LL tumor cells are heterogeneous with regards to their capability to recognize neoglycoproteins, as shown by fluorescence microscopy and flow cytofluorometry analyses; basically two distinct subpopulations could be evidenced which were called glucose-receptor-rich (or glucose-specific lectin-rich, GLR 3LL) and glucose-receptor-poor (or glucose-specific lectin-poor, GLP 3LL) cells; those two subpopulations could be separated on the basis of their binding properties to neoglycoprotein-substituted microcarriers onto which GLR 3LL cells were able to rapidly adhere (2 h) while GLP 3LL cells were not. "
04/01/2004 - "To evaluate a magnetic resonance (MR) imaging contrast agent for tumor detection based on paramagnetic nonionic vesicles (niosomes) bearing polyethylene glycol (PEG) and glucose conjugates for the targeting of overexpressed glucose receptors. "
09/01/1983 - "The aim of the present study was to find out whether the increase in plasma noradrenaline (NA) in response to exercise can be reduced by 1. increasing of the amount of carbohydrate available for metabolism without producing hyperinsulinemia and 2. by inhibition of afferent activity from hepatic glucoreceptors. "
01/01/1990 - "These results indicate that glucoreceptors that mediate hyperglycemia and a decrease in rGH in response to cerebral glucoprivation are located in the hindbrain."
07/24/1981 - "These results indicate that glucoreceptors that mediate feeding and hyperglycemia in response to cerebral glucoprivation are located in the caudal hindbrain and not in the hypothalamus where they have previously been sought."
11/01/1989 - "While peripheral administration of alloxan is a potent pancreatic beta-cell toxin, intracerebroventricular (ICV) alloxan reduces glucoprivic feeding in the absence of hyperglycemia, presumably through interactions with specific brain glucoreceptors. "
|4.||Growth Hormone (Somatotropin)
|6.||MM Form Creatine Kinase
|7.||Glutamic Acid (Glutamate)
|9.||Somatostatin (Somatotropin Release-Inhibiting Factor)