prostaglandin Bx

01/01/1982 - "PGBx, a polymeric prostaglandin, in chronic doses therapeutic for hereditary diabetes and hereditary obesity in mice, produces significant increases of blood neutrophils and lymphocytes in normal mice, hereditary diabetic mice, and hereditary obese mice. "
01/01/1980 - "This suggests that PGBx effects on obesity and appetite are not mediated through a blood glucose mechanism."
01/01/1980 - "Dose-dependent reduction of hereditary obesity in the non-diabetic mouse by polymeric prostaglandin PGBx."
01/01/1979 - "A polymeric derivative of prostaglandin B1, PGBX, which reactivates phosphorylation in damaged mitochondria, is shown to normalize the high blood glucose, obesity, and excessive appetite of the hereditary diabetic mouse. "

04/01/1987 - "These findings suggest that PGBx may be beneficial in the treatment of CNS ischemic injuries."

01/01/1980 - "Clearly PGBx radically improved cardiac recovery after circulatory arrest due to VF in the presence of acute myocardial infarction. "

08/01/1985 - "Preischemic perfusion with PGBx extended the time taken to reach 50% reduction in dP/dt following the first few seconds of ischemia. "
08/01/1985 - "PGBx had no effect on the development of intracellular acidosis during ischemia. "
01/01/1980 - "Myocardial recovery after ischemia: in vivo effects of PGBX, a polymeric derivative of PGB1."
01/01/1979 - "PGBx, a new polymeric derivative of PGB1, previously was shown to (a) restore oxidative phosphorylation to degraded isolated rat liver mitochondria in vitro and (b) to reverse the effects of cardiogenic ischemia in monkeys and cerebral ischemia in rabbits. "
08/01/1985 - "A polymeric prostaglandin (PGBx) attenuates adenine nucleotide loss during global ischemia and improves myocardial function during reperfusion."

07/01/1986 - "[Effects of polymeric prostaglandin PGBx in cerebral ischemia]."
01/01/1979 - "PGBx, a new polymeric derivative of PGB1, previously was shown to (a) restore oxidative phosphorylation to degraded isolated rat liver mitochondria in vitro and (b) to reverse the effects of cardiogenic ischemia in monkeys and cerebral ischemia in rabbits. "