|1.||Csillik, Bertalan: 3 articles (05/2010 - 01/2007)|
|2.||Knyihár-Csillik, Elizabeth: 2 articles (05/2010 - 01/2008)|
|3.||Mihály, András: 2 articles (05/2010 - 01/2008)|
|4.||Farkas, Ibolya: 2 articles (01/2008 - 01/2007)|
|5.||Herrala, Annakaisa M: 1 article (06/2014)|
|6.||Araujo, César L: 1 article (06/2014)|
|7.||Pulkka, Anitta E: 1 article (06/2014)|
|8.||Vihko, Pirkko: 1 article (06/2014)|
|9.||Kipar, Anja: 1 article (06/2014)|
|10.||Quintero, Ileana B: 1 article (06/2014)|
01/01/1989 - "Transganglionic degenerative atrophy, induced by a crush injury of the sciatic nerve, results in partial depletion of thiamine monophosphatase from ipsilateral segments L2-S1. "
01/01/1989 - "Reorganization of synaptic circuitry has been studied in the upper dorsal horn (Lamina II, substantia gelatinosa Rolandi) of the lumbar spinal cord in the adult rat, by means of electron histochemical visualization of thiamine monophosphatase after transganglionic degenerative atrophy. "
01/01/1988 - "Both of the axolemmal carbohydrate moieties were shown to be subjected to transganglionic regulation, even though the effects of transganglionic degenerative atrophy become evident considerably later than the depletion of axoplasmic marker substances like fluoride resistant acid phosphatase and thiamine monophosphatase."
01/01/2007 - "Blockade of retrograde transport of NGF results in transganglionic degenerative atrophy (TDA) in the segmentally related ipsilateral superficial spinal dorsal horn, which is characterized by depletion of the marker enzymes fluoride-resistant acid phosphatase (FRAP) and thiamine monophosphatase (TMP). "
01/01/1987 - "Whenever retrograde transport is impaired (either by injury or by blockade induced by perineural application of microtubule inhibitors) central terminals undergo transganglionic degenerative atrophy (TDA), characterized by depletion of substance P, somatostatin, FRAP (fluoride resistant acid phosphatase), TMPase (thiamine monophosphatase) and lectin-binding fucose-terminated glyco-conjugates. "
10/01/1981 - "The substantia gelationsa has been noted as a region of central pain control, In 1979, we reported that an intense thiamine monophosphatase (TMPase) activity was specifically localized on the plasma membrane of the synaptic glomeruli, especially dense sinusoid axon terminals (DSA). "
01/01/1991 - "In an effort to clarify the mechanism of pain accompanying thiamine deficiency, thiamine monophosphatase (TMPase) activity was demonstrated, by means of electron microscopic cytochemistry, in small ganglion cells in pyrithiamine-treated thiamine-deficient rats. "
01/01/2008 - "TDA induced by vinpocetine has been demonstrated to be followed by depletion of the marker enzyme fluoride-resistant acid phosphatase (FRAP) and its isoenzyme thiamine monophosphatase (TMP), and by the decrease in the pain-related neuropeptide substance P from laminae I-II-(III) from the segmentally related, ipsilateral substance of Rolando of the spinal cord. "
05/30/2010 - "Blockade of NGF transport is followed by transganglionic degenerative atrophy in the segmentally related, ipsilateral superficial spinal dorsal horn, characterized by depletion of the marker enzymes of nociception, fluoride resistant acid phosphatase (FRAP) and thiamine monophosphatase (TMP) from the Rolando substance and by decrease of the pain-related neuropeptides substance P (SP) and calcitonin gene-related peptide (CGRP) from lamina I-II-III. Based upon these findings, it has been suggested that vinpocetine may result in a locally restricted decrease of nociception. "
|3.||Ganglion Cysts (Ganglion)
|5.||Neuralgia (Stump Neuralgia)
|3.||Thymidine Monophosphate (TMP)
|5.||Nerve Growth Factor (NGF)
|9.||Calcitonin Gene-Related Peptide