|1.||Insel, Paul A: 2 articles (03/2007 - 04/2006)|
|2.||Arthur, David B: 2 articles (03/2007 - 04/2006)|
|3.||Paul, Richard J: 2 articles (04/2006 - 02/2005)|
|4.||Ishida, Yukisato: 2 articles (04/2006 - 02/2005)|
|5.||Gu, Min: 2 articles (04/2006 - 02/2005)|
|6.||Wardle, Robert L: 2 articles (04/2006 - 02/2005)|
|7.||Sun, Chi-Chin: 1 article (06/2009)|
|8.||Yang, Chuen-Mao: 1 article (06/2009)|
|9.||Wang, Wei-Jung: 1 article (06/2009)|
|10.||Wang, Hui-Hsin: 1 article (06/2009)|
|2.||Contact Dermatitis (Eczema, Contact)
01/01/1994 - "In clonal rat pheochromocytoma cells (PC12) we determined the kinetic parameters, Km and Vmax, of noradrenaline transport in the absence and presence of ATP gamma S. "
03/01/2007 - "Nerve growth factor (NGF)-promoted differentiation of pheochromocytoma 12 (PC12) cells enhanced the ability of the non-hydrolyzable ATP analog, ATPgammaS, to stimulate catecholamine (norepinephrine, NE) release and this enhancement occurred without a significant alteration in NE uptake. "
04/05/2006 - "We demonstrate here a previously unappreciated role for extracellular nucleotide signaling through the P2Y2 receptor in the survival of neurons: PC12 (pheochromocytoma 12) cells and dorsal root ganglion neurons are protected from serum starvation-induced apoptosis by ATP, UTP, and ATPgammaS, an effect mediated via P2Y2 receptors, as demonstrated by small interfering RNA and genetic knock-out models. "
04/01/2006 - "alpha-Toxin-permeabilized PCASM pretreated with ATPgammaS did not relax in response to hypoxia. "
04/01/2006 - "Moreover, when MRLC but not MYPT1 was protected from ATPgammaS thiophosphorylation by the MRLC kinase inhibitor ML7 (300 mum), hypoxia remained ineffective. "
02/01/2005 - "Hypoxia also relaxed GTPgammaS contractures but importantly, arteries could not be relaxed after treatment with ATPgammaS. "
08/01/2006 - "ATP released after hypotonic stress (200 mOsm/L) as well as P2YR agonists prevented hepatocyte killing by hypoxia with efficiency ranking UTP > ATPgammaS > ADPbetaS, whereas the P2XR agonist, methylene-adenosine-5'-triphosphate, was ineffective. "
08/01/2001 - "We found extracellular ATP and ATPgammaS, two P2 agonists, decreased osmotic fragility, enhanced cell volume recovery in response to hypotonic shock, and increased whole-cell currents. "
04/01/1993 - "Furthermore, the protein complexes of the M(r) 73,000 heat shock cognate protein and pRb110 were dissociated with the presence of ATP, but not with ADP and the nonhydrolyzable ATP analogue, ATP gamma S. "
|1.||adenosine 5'-O-(2-thiodiphosphate) (ADP-S)
|2.||2-Chloroadenosine (2 Chloroadenosine)
|3.||Adenosine Triphosphate (ATP)
|4.||Uridine Triphosphate (UTP)
|7.||tranilast (N 5')
|9.||Purinergic P2X2 Receptors
|10.||Purinergic P2Y2 Receptors