|1.||Lara, Lucienne S: 2 articles (01/2011 - 03/2010)|
|2.||Bełtowski, Jerzy: 2 articles (04/2007 - 01/2004)|
|3.||Wójcicka, Grazyna: 2 articles (04/2007 - 01/2004)|
|4.||Arcaya, José L: 1 article (10/2012)|
|5.||Rodríguez-Iturbe, Bernardo: 1 article (10/2012)|
|6.||Chávez, Maribel: 1 article (10/2012)|
|7.||Rincón, Jaimar: 1 article (10/2012)|
|8.||Pérez, Mariela: 1 article (10/2012)|
|9.||Romero-Vásquez, Freddy: 1 article (10/2012)|
|10.||García, Alberto J: 1 article (10/2012)|
|2.||Brain Hypoxia (Cerebral Hypoxia)
02/01/1982 - "Biochemical studies in rats revealed changes of (K+-Na+) ATPase activity, degree of oxidation and phosphorylation as well as dynamics of cAMP-cGMP and prostaglandin E, F2 alpha concentration in brain tissue in different period after cervical desympathization in conditions of normal cerebral blood supply and after circulatory cerebral hypoxia. "
|3.||Hypertension (High Blood Pressure)
10/01/2012 - "Overexpression of HGF transgene attenuates renal inflammatory mediators, Na(+)-ATPase activity and hypertension in spontaneously hypertensive rats."
06/01/2012 - "Finally, experimental data obtained using spontaneously hypertensive rats suggest that the Na(+)-ATPase could play a role in the pathogenesis of essential hypertension. "
03/01/2010 - "Na(+)-ATPase in spontaneous hypertensive rats: possible AT(1) receptor target in the development of hypertension."
10/01/2012 - "hHGF gene therapy normalized renal NF-κB activity, proinflammatory cytokines, antioxidant status (GSH, SOD and CAT), Na(+)-ATPase activity, reduced renal injury and ameliorated hypertension. "
04/21/2008 - "Causal relationship between sodium and hypertension has been proposed and various changes in Na+,K+-ATPase (sodium pump) activity have been described in established primary hypertension. "
12/01/2002 - "The response of four upregulated genes, ENA1 (encoding a Na+-ATPase also induced by saline stress) and PHO84, PHO89 and PHO12 (encoding genes upregulated by phosphate starvation), was characterized further. "
04/01/2007 - "Among the genes significantly up-regulated by Mg(2+) starvation, Ca(2+) stress, and alkalinization are ENA1 (encoding a P-type ATPase sodium pump) and PHO89 (encoding a sodium/phosphate cotransporter). "
01/01/2004 - "In rats with dietary-induced obesity, the Na(+),K(+)- ATPase activity was higher in the renal cortex and medulla by 19.7% and 34.3%, respectively, but Na(+)-ATPase was not different from control. "
01/01/2004 - "Spectrophotometric assay of renal ouabain-resistant Na(+)-ATPase and its regulation by leptin and dietary-induced obesity."
04/01/2007 - "Regulation of renal ouabain-resistant Na+-ATPase by leptin, nitric oxide, reactive oxygen species, and cyclic nucleotides: implications for obesity-associated hypertension."
|4.||Reactive Oxygen Species (Oxygen Radicals)
|6.||Ouabain (G Strophanthin)
|7.||Adenosine Triphosphate (ATP)
|8.||Sodium-Phosphate Cotransporter Proteins (Sodium-Phosphate Cotransporter)
|9.||GTP-Binding Proteins (G-Protein)
|10.||Myosin Heavy Chains (Myosin Heavy Chain)