|1.||Martin, Sarah: 1 article (10/2012)|
|2.||Yu, Jorn Cc: 1 article (10/2012)|
|3.||Petrikovics, Ilona: 1 article (10/2012)|
|4.||Stafford, Katelyn: 1 article (10/2012)|
|5.||Nasr, Jessica: 1 article (10/2012)|
|6.||Thompson, David: 1 article (10/2012)|
01/01/1995 - "Seizures and selective CA-1 hippocampal lesions induced by an excitotoxic cyanide metabolite, 2-iminothiazolidine-4-carboxylic acid."
01/01/1997 - "2-Iminothiazolidine-4-carboxylic acid produces hippocampal CA1 lesions independent of seizure excitation and glutamate receptor activation."
01/01/1997 - "We previously demonstrated that 2-iminothiazolidine-4-carboxylic acid (2-ICA), formed by cyanide reacting with cysteine, caused glutamate antagonist-sensitive seizures when injected i.c.v. (intracerebroventricular) in mice and produced hippocampal CA1 damage following i.c.v. infusion in rats. "
|1.||Glutamic Acid (Glutamate)
|2.||Glutamate Receptors (Glutamate Receptor)
|4.||arginine thiazolidinecarboxylate (ATCA)