|1.||Park, Jeen-Woo: 2 articles (11/2005 - 10/2003)|
|2.||Kim, Hyun Jeong: 1 article (11/2005)|
|3.||Choi, In Youl: 1 article (10/2003)|
|4.||Jung, Un Ju: 1 article (01/2003)|
|5.||Park, Yong Bok: 1 article (01/2003)|
|6.||Kwon, Oh-Shin: 1 article (01/2003)|
|7.||Choi, Myung-Sook: 1 article (01/2003)|
|8.||Lee, Mi-Kyung: 1 article (01/2003)|
|9.||Huh, Tae-Lin: 1 article (01/2003)|
11/18/2005 - "Upon exposure to heat shock, there was a distinct difference between the control cells and the cells pre-treated with 3mM oxalomalate for 3h in regard to apoptotic parameters, cellular redox status, and mitochondrial function. "
11/18/2005 - "The protective role of ICDH against heat shock-induced apoptosis in U937 cells was investigated in the control and the cells pre-treated with oxalomalate, a competitive inhibitor of ICDH. "
10/01/2003 - "Upon exposure to heat shock, the viability was lower and the protein oxidation, lipid peroxidation and oxidative DNA damage were higher in oxalomalate-treated cells as compared to control cells. "
11/18/2005 - "The oxalomalate pre-treated cells showed significant enhancement of apoptotic features such as activation of caspase-3, up-regulation of Bax, and down-regulation of Bcl-2 compared to the control cells upon exposure to heat shock. "
11/18/2005 - "Oxalomalate, a competitive inhibitor of NADP+-dependent isocitrate dehydrogenase, regulates heat shock-induced apoptosis."
|2.||Body Weight (Weight, Body)
01/01/2003 - "Oxalomalate did not lower the body weight and adipose tissue weight significantly; however, it significantly lower the plasma leptin concentration (p < 0.000), plasma and hepatic triglyceride levels (p < 0.01, p < 0.05), and adipocyte lipoprotein lipase activity (p < 0.01) compared to the control group. "
|2.||Isocitrate Dehydrogenase (Isocitrate Dehydrogenase (NAD+))
|3.||Caspase 3 (Caspase-3)
|5.||Lipoprotein Lipase (Diacylglycerol Lipase)