|1.||Dave, Alankruta R: 1 article (07/2014)|
|2.||Pandya, Megha G: 1 article (07/2014)|
|3.||Funyu, Shino: 1 article (01/2009)|
|4.||Amano, Yukinori: 1 article (01/2009)|
|5.||Suzuki, Ai: 1 article (01/2009)|
|6.||Wato, Eiji: 1 article (01/2009)|
|7.||Asahiyama, Masato: 1 article (01/2009)|
|8.||Lee, Hwi-Cheul: 1 article (06/2006)|
|9.||Nishihara, Masugi: 1 article (06/2006)|
|10.||Yamanouchi, Keitaro: 1 article (06/2006)|
|1.||Body Weight (Weight, Body)
03/01/2003 - "A dose-range finding study followed by a dose-response/effect study in Wistar rats investigated whether pre- and postnatal DEHA doses of 0, 800, or 1200mg/kg/day body weight and doses of 0, 200, 400, or 800mg/kg/day (main study) elicited developmental toxicity including antiandrogenic effects. "
03/01/2003 - "DEHA also induced a permanent decrease in offspring body weight (800mg/kg/day). "
05/01/1996 - "The major effects of chronic dosing were mortality in female rats at 500 mg/kg and in male and female mice at 750 mg/kg, accompanied by reductions in body weight gain in rats at 150 and 500 mg/kg and in mice at 750 mg/kg. Direct comparison of any tumorogenic effects of 2EH given alone to female mice with those due to 2EH formed in vivo from DEHA, DEHP, or TEHP is limited by the high mortality caused by 2ER in female mice at equivalent doses of 2EH. "
03/01/1995 - "Based on statistics on dietary habits it is concluded that the retail packaging of small portions of cheese even in a 'low migration' PVC cling film may lead to consumer intakes of DEHA close to or above the tolerable daily intake of 0.3 mg/kg body weight as defined by the EEC Scientific Committee for Food. "
02/01/1984 - "This is particularly relevant to assessments of the risk posed by DEHP and DEHA present as contaminants in foods, since human exposure via the food chain has been estimated by Shiota, Chou & Nishimura (1980) as 30 micrograms/kg body weight/day, several orders of magnitude less than the lowest exposure level used in these experiments. "
01/01/1984 - "Treatment with DEHA produced no significant effect on the lung tumor incidence of either sex. "
01/01/1986 - "Concern over the mutagenic and carcinogenic potential of these materials was stimulated by the finding that one member of this class, di-(2-ethylhexyl) adipate (DEHA), induced liver tumors in female mice in a chronic feeding study. "
11/01/1985 - "DEHA and DEHP also induced hepatocellular neoplasms in male mice, while DEHP caused hepatocellular neoplasms in both male and female rats. "
05/01/1984 - "It was the aim of this investigation to determine whether covalent binding of di(2-ethylhexyl) phthalate (DEHP) to rat liver DNA and of di(2-ethylhexyl) adipate (DEHA) to mouse liver DNA could be a mechanism of action contributing to the observed induction of liver tumors after lifetime feeding of the respective rodent species with high doses of DEHP and DEHA. "
|3.||Follicular Cyst (Follicular Cysts)
|1.||adipic acid (adipate)
|2.||Dimethyl Sulfoxide (DMSO)
|3.||Corn Oil (Oil, Corn)
|4.||4-bromobenzoyl-(alpha-aminoisobutyric acid)(5)-leucyl-(alpha-aminoisobutyric acid)(2)-methoxy (BALA)
|5.||Diethylhexyl Phthalate (DEHP)
|8.||Polyvinyl Chloride (PVC)
|9.||DNA (Deoxyribonucleic Acid)
|10.||phthalic acid (phthalate)