|1.||Effros, Richard M: 1 article (12/2011)|
|2.||Riezler, R: 1 article (08/2000)|
|3.||Spengler, U: 1 article (08/2000)|
|4.||Stabler, S P: 1 article (08/2000)|
|5.||Brensing, K A: 1 article (08/2000)|
|6.||Reichel, C: 1 article (08/2000)|
|7.||Rockstroh, J K: 1 article (08/2000)|
|8.||Sauerbruch, T: 1 article (08/2000)|
|9.||Berthold, H K: 1 article (08/2000)|
|10.||Look, M P: 1 article (08/2000)|
|1.||Decompression Sickness (Bends)
01/01/1995 - "The results obtained confirm the conclusions from previous studies, namely that beta-bends and 3(10)-helices are preferentially adopted by isovaline-rich peptides, a clear indication that this C alpha, alpha-disubstituted glycine tends to induce folded structures much more extensively than its unmethylated parent compound alpha-aminobutyric acid. "
01/01/1994 - "2. The effective doses of these compounds were lower than those required to prevent seizures due to NMDA in naive animals; these were in turn lower than those needed to prevent the convulsive effects of the alpha-aminobutyric acid (GABA) antagonist, bicuculline. "
11/01/1989 - "Moreover, alpha-methyltyrosine, and alpha-methyl-m-tyrosine also lowered metrazol threshold seizures, but had no effect on brain alpha-aminobutyric acid contents. "
11/01/1989 - "Furthermore, neither 5-hydroxytryptophan nor tranylcypromine had a significant effect on metrazol threshold seizures or brain alpha-aminobutyric acid contents, but blocked the changes previously induced by p-chlorophenyl alanine and reserpine."
01/01/1981 - "Intracellular concentrations of phenylalanine, tyrosine and alpha-aminobutyric acid in 13 homozygotes and 19 heterozygotes for phenylketonuria compared with 26 normals."
01/01/1980 - "Intracellular concentrations of phenylalanine, tyrosine and alpha-aminobutyric acid in 13 homozygotes and 19 heterozygotes for phenylketonuria (PKU) compared with 26 normals."
04/01/1996 - "In membranes of clone D2 cells the opioid peptide [D-Ala(2)]-leucine enkephalin (DADLE) produced a robust, concentration-dependent, stimulation of basal high-affinity GTPase activity; the prototypic opioid antagonist naloxone and the highly selective and potent delta opioid ligands H-Tyr-Tic-Phe-Phe-OH (TIPP) and H-Tyr-Tic[Ch2-NH]Phe-Phe-OH (TIPP[psi]) had little effect but N,N-diallyl-Tyr-Aib-Aib-Phe-Leu (ICI174864) caused a marked dose-dependent inhibition of this activity (Tic, 1,2,3,4-tetrahydroisoquinolin-2-yl-carbonyl]; Aib, alpha-aminobutyric acid). "
|5.||Ventricular Premature Complexes (Premature Ventricular Contraction)
08/01/1998 - "Bicuculline, a alpha-aminobutyric acid (GABA(A)) receptor antagonist, was administered intracisternally in pentobarbitone anaesthetized rabbits; 10 microg/kg intracisternal bicuculline induced polymorphic ventricular ectopic beats and ventricular tachycardia, while blood pressure increased by about 50-60% and heart rate in sinus rhythm decreased by about 20%. "
|2.||Glycine (Aminoacetic Acid)
|8.||GABA-A Receptor Antagonists
|9.||GTP Phosphohydrolases (GTPases)