|1.||Ganjam, Goutham K: 1 article (02/2005)|
|2.||Bahadur, Urvashi: 1 article (02/2005)|
|3.||Kondaiah, Paturu: 1 article (02/2005)|
|4.||Vasudevan, Nandini: 1 article (02/2005)|
|5.||Longcope, C: 1 article (08/2001)|
|6.||Browne-Martin, K: 1 article (08/2001)|
|7.||Bahadur, U: 1 article (03/2001)|
|8.||Vasudevan, N: 1 article (03/2001)|
|9.||Kondaiah, P: 1 article (03/2001)|
04/01/1984 - "At a daily dose of 24 micrograms for a period of 4 weeks RU 16117 led to 65% reduction in the number of already-established tumors. "
07/01/1982 - "Moxestrol showed a greater uptake selectivity in the tumors compared with ethynylestradiol. "
03/01/1977 - " It was considered likely that RU 16117 exerts its inhibitory activity at both the hypothalamic-pituitary and tumor levels."
03/01/1977 - " 8 and 24 mcg doses of RU 16117 led to 45 and 65% inhibition of tumor number, respectively, and tumor size was markedly reduced. "
03/01/1977 - "Not only the tumor number but also the tumor size was reduced by RU 16117 in a manner similar to that seen after ovariectomy. "
09/01/1985 - "Based on previous studies of the properties of moxestrol, we hypothesized that a radiohalogenated analog of moxestrol, [125I]11 beta-methoxy-17 alpha-iodovinyl-estradiol [( 125I] MIVE2), should bind to the estrogen receptor (ER) in some ovarian adenocarcinomas (OVCA), thereby offering the potential for imaging and/or treatment of these cancers. "
|4.||Hepatocellular Carcinoma (Hepatoma)
02/28/2005 - "Using moxestrol, an analogue of estrogen and transient transfection of deletion and mutation containing RCP promoter/reporter constructs in chicken hepatoma (LMH2A) cells, we identified an estrogen response unit (ERU) composed of two consensus ERE 1/2 sites and one non-consensus ERE 1/2 site. "
03/01/2001 - "Transient transfection of these RCP promoter deletion constructs into a chicken hepatoma cell line (LMH2A) showed 6-12-fold transcriptional induction by a stable estrogen analogue, moxesterol. "
08/01/2001 - "Our purpose was to examine the roles of natural (estradiol (E2) and estrone (E1)) and synthetic estrogens (ethinyl estradiol (EE), moxestrol (MOX), and tamoxifene (TAM)) in regulating production of sex hormone-binding globulin (SHBG) by human hepatoma G2 (Hep G2) cells, the rationale being that synthetic estrogens are less rapidly metabolized than natural estrogens and, thus, may alter SHBG levels more readily. "
09/01/1986 - "Oestradiol-17 beta, moxestrol, 2-fluoro-oestradiol and 4-fluoro-oestradiol all elicited pronounced LH surges and facilitated progesterone-triggered proceptive and lordosis behaviours.(ABSTRACT TRUNCATED AT 250 WORDS)"
04/01/1978 - "Comparison of the effects of estradiol-17beta and the synthetic estrogen, RU-2858, on lordosis behavior in adult female rats and guinea pigs."
|2.||Ethinyl Estradiol (Estinyl)
|8.||Estradiol Congeners (Synthetic Estrogens)
|9.||Estrogen Receptor Modulators (Antiestrogen)