|1.||Janahmadi, Mahyar: 3 articles (01/2013 - 05/2009)|
|2.||Goudarzi, Iran: 2 articles (09/2010 - 05/2009)|
|3.||Behzadi, Gila: 2 articles (09/2010 - 05/2009)|
|4.||Sapolsky, R M: 2 articles (02/2002 - 03/2000)|
|5.||Silva, Wagner A B: 1 article (08/2015)|
|6.||Cavalcante, Leny A: 1 article (08/2015)|
|7.||Santos de Abreu, Isadora: 1 article (08/2015)|
|8.||Allodi, Silvana: 1 article (08/2015)|
|9.||Medina, Bianca N S P: 1 article (08/2015)|
|10.||da Fonseca, Rodrigo N: 1 article (08/2015)|
|1.||Cerebellar Ataxia (Dysmetria)
01/01/2012 - "The aim of this work was to investigate the potential therapeutic activity of HuUCBMCs in the 3-acetylpyridine (3-AP) rat model of cerebellar ataxia. "
01/01/2015 - "Neurotoxin 3-acetylpyridine (3-AP) was intraperitoneally injected in rats to induce cerebellar ataxia. "
01/01/2013 - "Intra-peritoneal administration of riluzole has been shown to preserve the membrane properties and firing characteristics of Purkinje neurons in a rat model of cerebellar ataxia induced by 3-acetylpyridine (3-AP). "
05/01/2009 - "Here, the in vivo neuroprotective effect of riluzole on the intrinsic activity of Purkinje cells (PCs) in a rat model of cerebellar ataxia induced by 3-acetylpyridine (3-AP) was studied. "
06/01/2003 - "The neuroprotective role of estradiol in the inferior olive was then assessed in a model of cerebellar ataxia, achieved by the ip administration of 3-acetylpyridine (3-AP). "
01/01/2011 - "Profound alterations in the intrinsic excitability of cerebellar Purkinje neurons following neurotoxin 3-acetylpyridine (3-AP)-induced ataxia in rat: new insights into the role of small conductance K+ channels."
02/19/1998 - "3-acetylpyridine markedly decreased the maximal height of vertical jump, accompanied by motor incoordination. "
09/10/2010 - "Here, electrophysiological and behavioural consequences of in vivo co-treatment with 4-AP against 3-acetylpyridine (3-AP)-induced ataxia in rats were assessed. "
10/01/2013 - "This study investigated whether varenicline and nicotine could attenuate the ataxia induced in rats following destruction of the olivocerebellar pathway by the neurotoxin 3-acetylpyridine (3-AP). "
|3.||Body Weight (Weight, Body)
04/01/1985 - "In young rats (21-23 days old), 3-acetylpyridine caused dose- and time-related deficits in behavior, as measured by a neurological scale and by "tight-rope" performance, loss of body weight, and decreased survival. "
10/01/1984 - "At both 20 days and 30 days of age, rats which had received 3-acetylpyridine showed lower values in body weight, myelin yield, cerebroside level and specific activity of brain 2',3'-cyclic nucleotide-3-phosphohydrolase, when compared with those of the controls. "
10/01/1983 - "Body weight, myelin yield, cerebroside level and specific activity of 2',3'-cyclic nucleotide-3'-phosphohydrolase in the brain decreased by administration of 3-acetylpyridine. "
01/01/1983 - "Young adult mice of the ddN-F26 strain were given a single intraperitoneal injection of 3-acetylpyridine (3-AP) at a dosage of 200 mg/kg body weight. "
11/26/1984 - "Inferior olivary neurons: 3-acetylpyridine effects on glucose consumption, axonal transport, electrical activity and harmaline-induced tremor."
09/30/1983 - "Destruction of the climbing fibers with 3-acetylpyridine effectively blocked harmine, but not chlordecone-induced tremor, suggesting that chlordecone does not act through this pathway."
01/01/1977 - "Harmaline increases cerebellar 3':5'-cyclic guanosine monophosphate (cGMP) content in a dose-related manner; this increase is prevented by a pretreatment with 3-acetylpyridine (3-AP) (0.66 mmol/kg) which destroys climbing fibers and inhibits harmaline-induced tremor. "
02/01/1998 - "Ablation of the inferior olive by 3-acetylpyridine abolished the myoclonic seizure. "
06/15/1987 - "We examined the effect of destruction of cerebellar climbing fibers, by systemic administration of 3-acetylpyridine (3AP) or electrothermic lesion of the inferior olive, on seizures produced by various chemical convulsants in rats. "
10/01/1990 - "Bilateral inferior olive lesions, produced by systemic administration of the neurotoxin 3-acetylpyridine (3AP) produce a proconvulsant state specific for strychnine-induced seizures and myoclonus. "
|6.||Pyruvic Acid (Pyruvate)
|9.||Niacin (Nicotinic Acid)