|1.||Mahale, S: 1 article (01/2015)|
|2.||Chaudhuri, B: 1 article (01/2015)|
|3.||Vishwakarma, R A: 1 article (01/2015)|
|4.||Manda, S: 1 article (01/2015)|
|5.||Joshi, P: 1 article (01/2015)|
|6.||Jenkins, P R: 1 article (01/2015)|
|7.||Bharate, S B: 1 article (01/2015)|
|8.||Mao, Jian: 1 article (02/2013)|
|9.||Qing, Hong: 1 article (02/2013)|
|10.||Ma, Hong: 1 article (02/2013)|
|1.||Parkinson Disease (Parkinson's Disease)
08/17/1998 - "1,2,3,4-Tetrahydro-beta-carboline (TH beta C) is an endogenous or environmental neurotoxic factor putatively involved in the development of Parkinson's disease (PD). "
08/17/1998 - "Studies on the interaction between 1,2,3,4-tetrahydro-beta-carboline and cigarette smoke: a potential mechanism of neuroprotection for Parkinson's disease."
02/09/2001 - "These results suggest that cigarette smoke may prevent the accumulation of these proneurotoxins in the brain, which corroborate the involvement of the reaction between both TIQ and THbetaC with some components of tobacco smoke as a neuroprotective mechanism for Parkinson's disease."
02/09/2001 - "1,2,3,4-Tetrahydroisoquinoline (TIQ) and 1,2,3,4-tetrahydro-beta-carboline (THbetaC) are two endogenous or exogenous dopaminergic proneurotoxicants supposedly involved in the etiology of Parkinson's disease. "
09/01/2007 - "Based on the fact that the cyclization of N-[(3S)]-1,2,3,4-tetrahydro-beta-carboline-3-carboxyl]-l-lysine in both of acetic acid aqueous (5%) and rat plasma gave the same product and the hypothesis that the cyclization product is antithrombotic active, we report the synthesis, in vitro anti-aggregation, and in vivo anti-thrombosis activity of 20 hexahydropyrazino[1',2':1,6]pyrido[3,4-b]indole-1,4-dions (5a-t) as potential anti-thrombosis agents in this study. "
01/01/1975 - "6-Methoxy-1, 2, 3, 4, -tetrahydro-beta-carboline (6-MeO-THBC) was tested for anticonvulsant properties against audiogenic seizures in DBA/2J and primed C57BL/6J mice (i.e., mice given a prior auditory exposure) and aginast electroconvulsive seizures in DBA/2J mice. "
01/01/1980 - "All of the drugs tested which blocked AGS, including 6-MeO-THbetaC, THbetaC, 5-Hydroxytryptophan, chlorimipramine and pargyline, have biochemical similarities suggesting that facilitating serotonin function may be responsible for seizure-attenuating effects."
|2.||Acetic Acid (Vinegar)
|6.||Serotonin (5 Hydroxytryptamine)
|7.||Pargyline (Pargyline Hydrochloride)
|8.||Monoamine Oxidase (MAO)
|9.||5-Hydroxytryptophan (5 Hydroxytryptophan)
|10.||DNA (Deoxyribonucleic Acid)