|1.||Stowe, David F: 16 articles (04/2008 - 01/2002)|
|2.||Riess, Matthias L: 14 articles (02/2015 - 07/2002)|
|3.||Yan, Min: 12 articles (10/2013 - 02/2008)|
|4.||Camara, Amadou K S: 11 articles (12/2005 - 07/2002)|
|5.||Novalija, Enis: 10 articles (08/2003 - 01/2002)|
|6.||Engelhard, Kristin: 8 articles (01/2012 - 10/2003)|
|7.||Werner, Christian: 8 articles (12/2011 - 10/2003)|
|8.||Furuya, Hitoshi: 7 articles (04/2015 - 02/2002)|
|9.||Xiong, Lize: 7 articles (09/2014 - 10/2009)|
|10.||Wang, Qiang: 7 articles (09/2014 - 07/2008)|
07/01/2009 - "Rats anesthetized with sevoflurane during ischemia showed a significantly better outcome. "
06/01/2009 - "Sevoflurane preconditioning significantly improved functional outcome and reduced infarct volume (109 +/- 43 vs. 148 +/- 56 mm(3)) 3 days after ischemia compared to the control group. "
08/01/2007 - "Sevoflurane postconditioning is as effective as preconditioning in protecting myocardial function after global ischemia. "
04/21/2009 - "To investigate the protective effects of postconditioning with different concentrations of sevoflurane on renal ischemia-reperfusion (I/R) injury. "
05/01/2012 - "However, it had no effect on infarct size after 60 min ischemia (64.1 ± 5.9%, 62.3 ± 7.6% vs. 72.7 ± 9.2% for 2% sev, 4% sev vs. control, respectively, P > 0.05).The efficacy of sevoflurane postconditioning gradually weakened with increasing ischemia duration and disappears after 60 min ischemia in rabbits in vivo."
06/01/2012 - "Sevoflurane postconditioning has been proven to protect the hearts against ischemia/reperfusion injury, manifested mainly by improved cardiac function, reduced myocardial specific biomarker release, and decreased infarct size. "
05/01/2012 - "This experiment was designed to investigate the efficacy of sevoflurane postconditioning against ischemia-reperfusion injury. "
02/01/2007 - "The authors tested whether inhalation of sevoflurane at subanesthetic concentrations provides protection against postocclusive endothelial dysfunction induced by ischemia-reperfusion injury of the forearm in humans. "
02/01/2007 - "Sevoflurane inhalation at sedative concentrations provides endothelial protection against ischemia-reperfusion injury in humans."
12/01/2001 - "One MAC of sevoflurane provides protection against reperfusion injury in the rat heart in vivo."
|3.||Brain Ischemia (Cerebral Ischemia)
04/01/2012 - "Sevoflurane preconditioning produced marked improvement neurological functions and a reduction in brain infarct volumes than animals with brain ischemia only. "
01/01/2011 - "In conclusion, repeated sevoflurane preconditioning confered potent protection against brain ischemia, partly by improving BBB integrity."
08/31/2013 - "In this study, we explored the effect of sevoflurane conditioning (at concentrations of 1.5%, 2.4% or 3.0%) on the morphology and molecular mechanisms of the hippocampal CA1 region in male Sprague-Dawley rats subjected to global cerebral ischemia. "
08/15/2013 - "In the present study, the rats were exposed to sevoflurane for 15, 30, 60, and 120 minutes, followed by induction of permanent cerebral ischemia. "
01/01/2013 - "The aim of this study was to determine whether a combination of sevoflurane postconditioning and albumin provides additive neuroprotective effects after transient global cerebral ischemia in rats. "
|4.||Postoperative Nausea and Vomiting (PONV)
05/01/2003 - "In both studies, there was no difference in postdischarge outcomes at Day 7. Sevoflurane/sevoflurane was more costly with higher PONV rates in both studies. "
01/01/2011 - "Overall, the incidence of PONV was significantly lower in the TIVA and combined groups compared with the sevoflurane group (33.9%, 39.0% and 64.4%, respectively). "
08/01/2009 - "However, use of sevoflurane significantly increased both the risk of PONV and the number of postoperative nursing interventions. "
10/01/2008 - "Despite advantages of induction and maintenance of anaesthesia with sevoflurane, postoperative nausea and vomiting occurs frequently. "
09/01/2006 - "When compared to sevoflurane maintenance of anaesthesia, it allows for a more rapid recovery from anaesthesia as judged by recovery of saccadic eye movements and a decreased incidence of postoperative nausea and vomiting."
11/01/2009 - "We found that exposure of 3% sevoflurane during early reperfusion significantly improved functional recovery (improved left ventricular developed pressure (LVDP), +/-dp/dt, CF, HR and reduced left ventricular end-diastolic pressure (LVEDP)), decreased myocardial infarct size and reduced LDH and CK-MB release, when compared with unprotected hearts. "
09/01/2012 - "1.0 MAC sevoflurane and 1.5 MAC sevoflurane improved cardiac pump function after reperfusion and reduced myocardial infarct size with the increased iNOS expression (P<0.05). "
12/01/2015 - "The findings of the present study suggest that sevoflurane post-conditioning protects the myocardium against I/R injury and reduces the myocardial infarct size. "
11/01/2014 - "Sevoflurane in acute myocardial infarction: a pilot randomized study."
11/01/2014 - "Corrected Thrombolysis In Myocardial Infarction frame count was 12.3 ± 1.5 in the sevoflurane group and 15.6 ± 9.1 in the control group (P = .16). "
|7.||desflurane (I 653)
|8.||Adenosine Triphosphate (ATP)