|1.||Colgan, Sean P: 2 articles (03/2008 - 10/2002)|
|2.||Stanisławowski, Marcin: 1 article (12/2015)|
|3.||Bigda, Jacek: 1 article (12/2015)|
|4.||Koszałka, Patrycja: 1 article (12/2015)|
|5.||Majewski, Marceli: 1 article (12/2015)|
|6.||Wierzbicki, Piotr: 1 article (12/2015)|
|7.||Stasiłojć, Grzegorz: 1 article (12/2015)|
|8.||Składanowski, Andrzej C: 1 article (12/2015)|
|9.||Urban, Aleksandra: 1 article (12/2015)|
|10.||Gołuńska, Monika: 1 article (12/2015)|
10/01/2002 - "In vivo studies in a murine hypoxia model revealed that hypoxia-induced CD73 may serve to protect the epithelial barrier, since the CD73 inhibitor alpha,beta-methylene ADP promotes increased intestinal permeability. "
08/01/1994 - "In addition, AOPCP decreased the basal work load and coronary flow of the heart, slightly attenuated the hypoxia-induced flow increase and prevented adenylate loss during hypoxia. "
03/01/1992 - "Coronary vascular resistance decreased during hypoxia but the decline in resistance was less in AOPCP treated hearts. "
03/01/1992 - "Epicardial and venous AMP levels were elevated by AOPCP treatment during normoxia and hypoxia. "
03/01/1992 - "Epicardial and venous adenosine levels increased during hypoxia with AOPCP but the increases were lower than those for untreated hypoxic hearts. "
10/01/1998 - "min-1, respectively); here plasma AOPCP levels were established before and during PC but not during the subsequent prolonged ischemia. "
11/01/2005 - "During 220 min of ischemia, extracellular ATP levels significantly increased 1.3-fold in AOPCP-treated rats (12.62+/-0.65 nM, p < 0.01 versus normoxic level). "
06/01/1999 - "AOPCP significantly increased dialysate AMP levels; at 60 minutes ischemia AMP levels were 6.22 +/- 2.97 microM in control hearts and 38.60 +/- 5.69 microM in AOPCP treated hearts. "
06/01/1999 - "At 30 minutes ischemia dialysate adenosine concentrations were 19.47 +/- 2.72, 11.41 +/- 2.44, and 7.93 +/- 1.01 microM in control, homocysteine, and AOPCP hearts, respectively. "
03/01/1994 - "To test the idea that the infarct size-limiting effect of ischemic preconditioning is blunted by inhibition of ectosolic 5'-nucleotidase activity, we assessed 5'-nucleotidase activity, adenosine release, and infarct size caused by sustained ischemia with and without an exposure to alpha,beta,-methylene adenosine 5'-diphosphate (AOPCP) in the ischemia-preconditioned myocardium. "
03/15/2008 - "Likewise, enteral administration of the selective CD73 inhibitor alpha,beta-methylene ADP to cd73(+/+) mice resulted in a similar increase in severity of TNBS colitis. "
02/01/2009 - "alpha,beta-Methylene ADP (100 microM), an inhibitor of CD39, restored ATP-induced vasoconstriction in arterioles from mice with DSS-induced colitis. "
06/01/2002 - "Anesthetized rabbits underwent 30 min coronary occlusion and 3 h reperfusion in ten groups: (1) Control, (2) Low Mg, (3) Mg, (4) High Mg, (5) calcium (Ca), (6) Mg+Ca, (7) 8-phenyltheophylline (8PT), an adenosine receptor blockade, (8) 8PT+Mg, (9) alpha, beta-methylene-adenosine diphosphate (AOPCP), a selective inhibitor of ecto-5'-nucleotidase, and (10) AOPCP+Mg groups. "
|5.||Ganglion Cysts (Ganglion)
08/01/1999 - "In the presence of AOPCP (alpha,beta-methyleneadenosine 5'-diphosphate), an ecto-5'-nucleotidase inhibitor, the ganglion-stimulated increase in interstitial ATP and adenosine reached levels similar to those in the absence of AOPCP, also suggesting that adenosine does not derive from extracellular ATP. "
|1.||5'-Nucleotidase (5' Nucleotidase)
|3.||Adenosine Triphosphate (ATP)
|4.||Adenosine Monophosphate (AMP)
|5.||Purinergic P1 Receptors (Adenosine Receptor)
|6.||5'-Guanylic Acid (GMP)
|8.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|9.||Nucleoside Transport Proteins (Nucleoside Transporter)
|10.||Tetraethylammonium (Tetraethylammonium Bromide)